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Murine Lyme borreliosis: route of inoculation determines immune response and infectivity

A R Pachner1, E Delaney, N S Ricalton

  • 1Georgetown University School of Medicine, Department of Neurology, Washington, DC 20007.

Regional Immunology
|November 1, 1992
PubMed

Insights

Outer surface protein A (OspA) from Borrelia burgdorferi, the Lyme disease agent, is a vaccine candidate. OspA immunoreactivity was induced in mice by various infection routes, except intradermal inoculation, suggesting skin infection alters OspA immunogenicity.

Area of Science:

  • Immunology
  • Microbiology
  • Vaccine Development

Background:

  • Outer surface protein A (OspA) is a key Borrelia burgdorferi antigen and a potential Lyme disease vaccine candidate.
  • OspA is recognized late in human Lyme disease but is immunogenic in experimental models.

Purpose of the Study:

  • To investigate the immunogenicity of OspA in mice following different infection routes and conditions.
  • To determine if the route of Borrelia burgdorferi infection influences the immune response to OspA.

Main Methods:

  • Mice were infected with Borrelia burgdorferi via intraperitoneal, intravenous, or intradermal routes.
  • OspA immunoreactivity was assessed using Western blotting.
  • Variables included inoculum size, spirochete isolate, and mouse strain.

Main Results:

  • OspA immunoreactivity was readily elicited by injecting spirochete sonicates.
  • Intraperitoneal or intravenous infection with infectious spirochetes induced OspA immunoreactivity.
  • Intradermal infection did not elicit significant OspA immunoreactivity, with responses directed against other B. burgdorferi proteins.

Conclusions:

  • The route of Borrelia burgdorferi infection significantly impacts OspA immunogenicity in mice.
  • Intradermal infection may lead to alterations in OspA, reducing its immunogenic potential.
  • These findings have implications for the efficacy of OspA-based Lyme disease vaccines.

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