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Fungus-specific translation elongation factor 3 gene present in Pneumocystis carinii

M F Ypma-Wong1, W A Fonzi, P S Sypherd

  • 1Department of Microbiology and Molecular Genetics, University of California, Irvine 92717.

Infection and Immunity
|October 1, 1992
PubMed

Insights

Pneumocystis carinii pneumonia (PCP) is a significant AIDS complication. Genetic analysis reveals Pneumocystis carinii possesses a fungal translation elongation factor 3 (EF-3), supporting its classification as a fungus.

Area of Science:

  • Mycology
  • Molecular Biology
  • Infectious Diseases

Background:

  • Pneumocystis carinii pneumonia (PCP) is a leading cause of illness and death in AIDS patients.
  • Antiprotozoan drugs are effective for PCP treatment and prevention, supporting its classification as a protozoan.
  • Recent genetic findings challenge the protozoan classification of P. carinii.

Purpose of the Study:

  • To investigate the classification of Pneumocystis carinii.
  • To identify fungal-specific genes within P. carinii.
  • To provide further evidence for the fungal nature of P. carinii.

Main Methods:

  • Genetic analysis of Pneumocystis carinii.
  • Isolation of the gene for translation elongation factor 3 (EF-3).
  • Comparison of EF-3 gene with those from other eukaryotes.

Main Results:

  • A gene for translation elongation factor 3 (EF-3) was identified and isolated from P. carinii.
  • EF-3 is a unique component of fungal protein synthesis.
  • The presence of EF-3 in P. carinii indicates a fungal classification.

Conclusions:

  • Pneumocystis carinii exhibits genetic characteristics consistent with fungi.
  • The identification of the EF-3 gene provides strong evidence for reclassifying P. carinii as a fungus.
  • This finding has implications for understanding PCP pathogenesis and treatment.

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