Related Experiment Videos

2-5A synthetase expression in mice infected with Trypanosoma cruzi

R Marcovistz1, D Decote Ricardo, S C Gonçalves da Costa

  • 1Departamento de Imunologia, Instituto Oswaldo Cruz, Fiocruz, Rio de Janeiro, Brasil.

Pathologie-Biologie
|March 1, 1992
PubMed

Insights

Colombian strain T. cruzi infection induces higher interferon levels and greater 2-5A synthetase activity in mice compared to Y strain. This indicates active interferon production and action in infected organs.

Area of Science:

  • Immunology
  • Parasitology
  • Molecular Biology

Background:

  • Interferon (IFN) plays a crucial role in antiviral and antiparasitic responses.
  • Trypanosoma cruzi (T. cruzi) infection, the causative agent of Chagas disease, can modulate the host immune system.
  • The specific role of IFN in T. cruzi infection and its strains is not fully elucidated.

Purpose of the Study:

  • To investigate the production and action of interferon in mice infected with two different strains of T. cruzi: Colombian and Y.
  • To compare the levels of interferon production and the activity of an interferon-mediated enzyme, 2-5A synthetase, between the two T. cruzi strains.

Main Methods:

  • Mice (Balb/c strain) were infected with Colombian or Y strain T. cruzi.
  • Interferon activity was measured in plasma and organ extracts (spleen, lung, heart) using in vitro assays.
  • The activity of 2-5A synthetase, an interferon-induced enzyme, was quantified in spleen, lung, and heart extracts at various time points post-infection.

Main Results:

  • Colombian strain T. cruzi infection resulted in significantly higher interferon levels compared to the Y strain under identical experimental conditions.
  • A corresponding greater induction of 2-5A synthetase activity was observed in mice infected with the Colombian strain.
  • Elevated levels of 2-5A synthetase were detected in multiple organs, indicating a systemic response to interferon.

Conclusions:

  • Interferon produced during T. cruzi infection in mice is biologically active.
  • The Colombian strain of T. cruzi elicits a stronger interferon response than the Y strain.
  • The findings suggest that interferon plays a role in the host's response to T. cruzi infection, with strain-dependent variations.

Related Concept Videos