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Hepatitis C virus infection in chronic liver disease in Bombay

D N Amarapurkar1, A Kumar, S S Parikh

  • 1Department of Gastroenterology, B Y L Nair Charitable Hospital, Bombay.

Insights

Hepatitis C virus (HCV) antibody prevalence was studied in 126 chronic liver disease patients in Bombay. The study found HCV present in 16.6% of patients, with higher rates in cryptogenic liver disease.

Area of Science:

  • Hepatology
  • Virology
  • Epidemiology

Background:

  • Chronic liver disease is a significant health concern.
  • Hepatitis C virus (HCV) is a major cause of chronic liver disease worldwide.
  • Understanding HCV prevalence in specific populations is crucial for public health strategies.

Purpose of the Study:

  • To determine the prevalence of antibody to Hepatitis C Virus (anti-HCV) among patients with chronic liver disease in Bombay.
  • To investigate the association of anti-HCV with other liver disease etiologies, including Hepatitis B Virus (HBV) and alcohol consumption.

Main Methods:

  • Sera from 126 patients with chronic liver disease (cirrhosis, cirrhosis with hepatocellular carcinoma, chronic active hepatitis) were tested.
  • Enzyme-Linked Immunosorbent Assay (ELISA) was used to detect Hepatitis B surface antigen (HBsAg) and anti-HCV antibody.
  • HBsAg-positive samples were further tested for anti-delta antibody and IgM anti-HBc.

Main Results:

  • Overall, 21 out of 126 patients (16.6%) tested positive for anti-HCV antibody.
  • The prevalence of anti-HCV was 13.7% in HBsAg-positive patients, 14.7% in alcoholic liver disease patients, and 20.5% in cryptogenic liver disease patients.
  • A history of blood transfusion was reported by 38% of anti-HCV positive patients.

Conclusions:

  • Hepatitis C virus infection is a notable factor in chronic liver disease patients in Bombay, with an estimated prevalence of 15-20%.
  • The study highlights the importance of considering HCV in the etiological diagnosis of chronic liver disease, particularly in cryptogenic cases.
  • Blood transfusion appears to be a significant risk factor for HCV acquisition in this population.

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