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Evidence for actinlike proteins in an M protein-negative strain of Streptococcus pyogenes

L A Barnett1, M W Cunningham

  • 1Department of Microbiology and Immunology, University of Oklahoma Health Sciences Center, Oklahoma City 73190.

Infection and Immunity
|September 1, 1992
PubMed

Insights

Streptococcus pyogenes may possess actin-like proteins, potentially explaining autoimmune reactions in rheumatic fever. These bacterial proteins mimic human actin, reacting with antibodies targeting heart tissue.

Area of Science:

  • Microbiology
  • Immunology
  • Biochemistry

Background:

  • Autoimmune cardiac injury in acute rheumatic fever is linked to shared antigens between Streptococcus pyogenes and heart tissue.
  • Cross-reactive antibodies in rheumatic fever target both S. pyogenes antigens (M protein, 60-kDa antigen) and heart components like myosin and actin.

Purpose of the Study:

  • To investigate the presence and characteristics of actin-like proteins in Streptococcus pyogenes.
  • To determine if S. pyogenes contains proteins that mimic eukaryotic actin and could contribute to autoimmune responses.

Main Methods:

  • Utilized established protocols for eukaryotic actin polymerization and isolation to extract proteins from S. pyogenes.
  • Employed immunoblots with antiactin monoclonal antibodies to detect actin-like proteins.
  • Assessed protein behavior using myosin and DNase I affinity chromatography.
  • Performed electron microscopy to visualize protein filaments.
  • Measured enhancement of eukaryotic myosin ATPase activity.

Main Results:

  • Two proteins of approximately 60 kDa in the polymerized bacterial extract reacted with an antiactin antibody.
  • Proteins of 43 kDa and 60 kDa from the extract exhibited actin-like behavior, binding to myosin and DNase I affinity columns.
  • Electron microscopy revealed filament formation in the bacterial extract.
  • The bacterial extract enhanced the ATPase activity of eukaryotic myosin.

Conclusions:

  • The study provides evidence for the presence of actin-like proteins in Streptococcus pyogenes.
  • These findings suggest a potential mechanism for molecular mimicry, where bacterial actin-like proteins could trigger autoimmune responses against heart tissue in rheumatic fever.

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