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Effects of macrolides on ultrastructure of Staphylococcus aureus during postantibiotic phase

T Watanabe1, M Kanno, E Tejima

  • 1Pharmaceutical Research Centre, Meiji Seika Kaisha, Ltd., Yokohama, Japan.

Drugs Under Experimental and Clinical Research
|January 1, 1992
PubMed

Insights

Midecamycin acetate demonstrated the longest postantibiotic effect (PAE) against Staphylococcus aureus, impacting bacterial cell wall structure. Other macrolides like josamycin and clarithromycin also showed significant PAEs and ultrastructural changes.

Area of Science:

  • Microbiology
  • Pharmacology
  • Cell Biology

Background:

  • Macrolide antibiotics are crucial for treating bacterial infections.
  • Understanding their postantibiotic effects (PAEs) and impact on bacterial ultrastructure is vital for optimizing therapy.
  • Staphylococcus aureus is a significant human pathogen requiring effective treatment strategies.

Purpose of the Study:

  • To investigate the postantibiotic effects (PAEs) of midecamycin acetate (MOM), erythromycin (EM), josamycin (JM), and clarithromycin (CAM) on Staphylococcus aureus.
  • To examine the ultrastructural changes in S. aureus during the PAE phase induced by these macrolides.
  • To compare the PAE durations and morphological alterations caused by different macrolide antibiotics.

Main Methods:

  • In vitro exposure of Staphylococcus aureus to 2x Minimum Inhibitory Concentration (MIC) of macrolides for 2 hours.
  • Measurement of postantibiotic effect (PAE) durations for each antibiotic.
  • Transmission electron microscopy (TEM) to analyze ultrastructural changes in bacterial cells during the PAE.
  • Evaluation of serum levels and corresponding PAEs in humans.

Main Results:

  • Midecamycin acetate (MOM) exhibited the longest PAE in vitro (3.9 h) and in human serum (2.4 h).
  • Josamycin (JM) and clarithromycin (CAM) showed moderate PAEs (in vitro: 2.5 h and 1.9 h; serum: 1.4 h and 1.3 h, respectively).
  • Erythromycin (EM) demonstrated a shorter PAE (in vitro: 1.2 h; serum: minimal effect).
  • Ultrastructural changes, including thickened cell walls and enlarged cell size (1.5-2x), were observed during the PAE phase for all tested macrolides, with MOM inducing changes for a longer duration (4 h).

Conclusions:

  • Midecamycin acetate possesses a potent and prolonged postantibiotic effect against Staphylococcus aureus, accompanied by significant and lasting ultrastructural alterations.
  • Macrolide antibiotics induce observable changes in Staphylococcus aureus ultrastructure during their respective PAE phases.
  • The findings suggest that midecamycin acetate may offer advantages in managing S. aureus infections due to its extended PAE and impact on bacterial morphology.

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