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Management of esophagogastric corrosive injuries in children
Insights
Caustic ingestion in children can lead to esophageal stenosis and gastric outlet obstruction. Balloon dilatation is a primary treatment for esophageal strictures, though it carries perforation risks.
Area of Science:
- Gastroenterology
- Pediatric Surgery
- Toxicology
Background:
- Caustic esophageal injuries are increasingly seen in pediatric populations.
- Ingestion of corrosive substances, particularly alkali, poses significant risks.
Purpose of the Study:
- To analyze complications and treatment outcomes for corrosive esophagogastric injuries in children.
- To evaluate the efficacy of different management strategies for esophageal strictures and gastric outlet obstruction.
Main Methods:
- Retrospective analysis of 120 pediatric caustic ingestion cases admitted between 1990 and 2000.
- Review of management strategies including dilatation, colonic interposition, and surgical interventions.
- Assessment of complications such as esophageal stenosis and gastric outlet obstruction.
Main Results:
- Esophageal stenosis occurred in 25.8% and gastric outlet obstruction in 5% of patients.
- Balloon dilatation was the primary treatment for esophageal strictures, with 4 perforations observed.
- Surgical interventions like colonic interposition and pyloroplasty were employed for refractory cases.
Conclusions:
- Balloon dilatation is a recommended first-line treatment for esophageal strictures despite perforation risks.
- Gastric outlet obstruction often necessitates surgical intervention after thorough endoscopic evaluation.
- Timely and appropriate management is crucial for improving outcomes in pediatric caustic ingestion injuries.
Abstract:
There has been an increase in the number of patients admitted to our hospital with caustic esophageal injuries during the last five years. The aim of this study was to analyze the complications and results of the treatment of corrosive esophagogastric injury. Between 1990 and 2000, 120 caustic ingestion accidents were admitted to our unit. The mean age was 4 years, with a 2 : 1 male to female ratio. The average time between the caustic ingestion and admission to hospital was 14.9 days. The ingested substances were alkali in 80.9 % and acid in 19.1 % of the cases. Stenosis of the esophagus developed in 31 (25.8 %) and gastric outlet obstruction (GOO) in 6 (5 %) patients. Management of the esophageal stricture consisted of dilatation in 28 patients. Three children underwent colonic interposition without a dilatation attempt. Six children were lost to follow-up; 4 patients were successfully treated; 13 patients were still in the dilatation program at the time of writing with 6 improving and 2 patients waiting for interposition surgery; 4 patients underwent colonic interposition and 1 patient underwent resection of the stenotic part of the esophagus. Among the patients in the dilation program, we observed 4 esophageal perforations. Three of them were treated medically and further dilatations were carried out, while one was managed by colonic interposition. The treatment modalities for GOO cases consisted of pyloroplasty in 3, Billroth I in 2 and balloon dilation of the pylorus in 1 child. Although balloon dilatation of the esophagus carries the risk of perforation, it should be the first line of treatment in suitable cases. GOO cases may require surgical therapy following a detailed endoscopic evaluation.
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