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The kallikrein-kinin system in early state of diabetes

S B Vila1, V A Peluffo, C Alvarado

  • 1Cátedra de Fisiología, Facultad de Farmacia y Bioquímica, Hospital General de Niños Dr. Pedro Elizalde, Buenos Aires, Argentina.

Agents and Actions. Supplements
|January 1, 1992
PubMed

Insights

Diabetic patients show elevated levels of total and pre-kallikrein, key components of the kallikrein-kinin system. These increases correlate with microalbuminuria, suggesting potential impacts on renal hemodynamics in diabetes.

Area of Science:

  • Biochemistry
  • Nephrology
  • Endocrinology

Background:

  • The kallikrein-kinin system plays a role in regulating blood pressure and kidney function.
  • Diabetic nephropathy is a common complication characterized by microalbuminuria.
  • Understanding the kallikrein-kinin system's involvement in diabetes is crucial for managing kidney complications.

Purpose of the Study:

  • To investigate the kallikrein-kinin system's activity in children with diabetes.
  • To determine the relationship between microalbuminuria and kallikrein-kinin system components in diabetic patients.
  • To explore potential links between kallikrein-kinin system alterations and renal hemodynamic changes in diabetes.

Main Methods:

  • Study included 9 healthy children and 15 diabetic children (ages 1-14 years).
  • Measured total and pre-kallikrein levels in participants.
  • Assessed microalbuminuria levels (mg/24 h) to categorize patients.

Main Results:

  • Diabetic patients exhibited increased total and pre-kallikrein compared to healthy controls.
  • Patients with low microalbuminuria showed 3-fold (total kallikrein) and 2-fold (pre-kallikrein) increases.
  • Patients with high microalbuminuria demonstrated over 4-fold (total kallikrein) and 8-fold (pre-kallikrein) increases.

Conclusions:

  • Total kallikrein and pre-kallikrein are elevated in diabetic individuals.
  • Higher microalbuminuria levels correlate with greater increases in total and pre-kallikrein.
  • These kallikrein-kinin system alterations may influence renal hemodynamics in diabetic patients.

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