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Delayed lactogenesis II: a comparison of four cases
Christine M Betzold1, Kathleen L Hoover, Cathy L Snyder
1LacNackrnp@aol.com
Journal of Midwifery & Women'S Health
|March 11, 2004
Summary
Delayed milk production, or lactogenesis II, can be caused by hormonal issues like gestational ovarian theca lutein cysts. Early detection via testosterone levels aids management, enabling successful breastfeeding for most affected mothers.
Area of Science:
- Reproductive Endocrinology
- Lactation Physiology
Background:
- Lactogenesis II, the onset of copious milk production, typically occurs 30-40 hours postpartum.
- Delayed lactogenesis II can stem from various hormonal and non-hormonal factors.
- Standard blood tests can help identify hormonal causes of delayed milk production.
Observation:
- Gestational ovarian theca lutein cysts are identified as a potential cause of delayed lactogenesis II.
- These cysts can be detected by measuring testosterone levels.
- The delay in milk production associated with these cysts can last up to 31 days.
Findings:
- Measuring testosterone levels can aid in diagnosing theca lutein cysts causing delayed lactogenesis II.
- Proper management strategies exist for women with this condition.
- Three out of four women in the study successfully established full breastfeeding despite the delay.
Implications:
- Early identification of hormonal imbalances, such as those caused by theca lutein cysts, is crucial for managing delayed lactogenesis II.
- Timely diagnosis and intervention can lead to successful breastfeeding outcomes.
- Further research into hormonal influences on lactation is warranted.