Patterns of collateral pathways in extrahepatic portal hypertension as demonstrated by multidetector row computed
Ihab R Kamel1, Leo P Lawler, Frank M Corl
1Russel H Morgan Department of Radiology, Johns Hopkins Hospital, Baltimore, MD 21287, USA. ikamel@hjmi.edu
Abstract:
Pancreatic neoplasm can compromise the portal vein or its tributaries, resulting in extrahepatic portal hypertension. Collateral channels may develop, usually in a hepatopetal direction. These include gastroepiploic veins, short gastric veins, left gastric vein, left colic veins, and spontaneous splenorenal shunts. We present the spectrum of these collaterals as delineated by multidetector row computed tomography with advanced image processing. Delineation of these collaterals is important before surgery so as to avoid possible uncontrollable bleeding if they are inadvertently disrupted.
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