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Phi 29 DNA polymerase active site. Mutants in conserved residues Tyr254 and Tyr390 are affected in dNTP binding

M A Blasco1, J M Lázaro, A Bernad

  • 1Centro de Biología Molecular, Universidad Autónoma, Canto Blanco, Madrid, Spain.

Insights

Investigating mutations in phi 29 DNA polymerase reveals that specific tyrosine residues are crucial for magnesium-dependent nucleotide binding and DNA replication. These findings shed light on the enzyme's active site and substrate interactions.

Area of Science:

  • Molecular Biology
  • Enzymology
  • Biochemistry

Background:

  • Phi 29 DNA polymerase, an alpha-like DNA polymerase, possesses conserved amino acid regions implicated in its polymerization active site.
  • Two key conserved regions, region 1 (motif "D-NSLYP") and region 2a (motif "K--NS(L/V)YG"), are critical for polymerase function.

Purpose of the Study:

  • To investigate the role of specific tyrosine residues (Tyr254 and Tyr390) in conserved regions 1 and 2a of phi 29 DNA polymerase.
  • To elucidate the involvement of these residues in magnesium-dependent nucleotide binding and DNA replication initiation.

Main Methods:

  • Site-directed mutagenesis was used to create phi 29 DNA polymerase mutants: Y254F, Y390F, and Y390S.
  • Enzyme activity assays were performed using magnesium-dNTPs and manganese-dNTPs as substrates.
  • Analysis of protein-primed initiation and template-independent deoxynucleotidylation was conducted.

Main Results:

  • Mutant polymerases showed impaired polymerization with Mg(2+)-dNTPs but normal activity with Mn(2+)-dNTPs.
  • The Y254F mutant exhibited reduced affinity for the initiating nucleotide (Mg(2+)-dATP) and altered dNTP affinity during initiation.
  • Mutants Y390F and Y390S displayed hypersensitivity to specific dNTP analogs.

Conclusions:

  • Residues Tyr254 and Tyr390 are essential for Mg(2+)-dNTP binding in phi 29 DNA polymerase.
  • These findings highlight the direct or indirect involvement of these tyrosine residues in substrate recognition and catalytic activity.
  • The study provides insights into the mechanism of alpha-like DNA polymerases and their active site structure.

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