p53 upregulates death receptor 4 expression through an intronic p53 binding site
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Updated: Aug 23, 2026

Yeast As a Chassis for Developing Functional Assays to Study Human P53
Published on: August 4, 2019
Xiangguo Liu1, Ping Yue, Fadlo R Khuri
1Winship Cancer Institute, Emory University School of Medicine, 1365-C Clifton Road, Clinical Building C-3088, Atlanta, GA 30322, USA.
Abstract:
Death receptor 4 (DR4) is one of the tumor necrosis factor-related apoptosis-inducing ligand (TRAIL) receptors and triggers apoptosis on ligation with TRAIL or overexpression. Our previous study demonstrated that DR4 expression could be regulated in a p53-dependent fashion. In the present study, we have demonstrated that DR4 is a p53 target gene and is regulated by p53 through a functional intronic p53 binding site (p53BS) based on the following lines of evidence: (a) the p53BS in the DR4 gene is almost identical to the one found in the first intron of the DR5 gene in terms of their locations and sequences; (b) DR4 p53BS bound to p53 protein in intact cells upon p53 activation as demonstrated by a chromatin immunoprecipitation assay; (c) a luciferase reporter vector carrying the DR4 p53BS upstream of an SV40 promoter exhibited enhanced luciferase activity when transiently cotransfected with a wild-type p53 expression vector in p53-null cell lines or stimulated with DNA-damaging agents in a cell line having wild-type p53; and (d) when the DR4 p53BS, together with its own corresponding promoter region in the same orientation as it sits in its natural genomic locus, was cloned into a basic luciferase vector without a promoter element, its transcriptional activity was strikingly increased by cotransfection of a wild-type p53 expression vector or treatment with DNA-damaging agents. However, wild-type p53 or DNA-damaging agents completely lost their activity to increase transcriptional activity of a reporter construct with deleted DR4 p53BS. Thus, we conclude that p53 directly regulates the expression of the DR4 gene via the novel intronic p53BS.
Insights
The tumor suppressor p53 directly regulates Death Receptor 4 (DR4) gene expression. This regulation occurs through a specific binding site within the DR4 gene, confirming DR4 as a p53 target gene.
Area of Science:
- Molecular Biology
- Cancer Biology
- Genetics
Background:
- Death Receptor 4 (DR4) is a key mediator of apoptosis, activated by TRAIL.
- Previous research indicated a p53-dependent regulation of DR4 expression.
Purpose of the Study:
- To determine if DR4 is a direct p53 target gene.
- To identify the regulatory mechanism of p53 on DR4 expression.
Main Methods:
- Chromatin immunoprecipitation assays to assess p53 binding to the DR4 gene.
- Luciferase reporter assays to evaluate transcriptional activity regulated by the p53 binding site.
- Analysis of p53 binding site (p53BS) in the DR4 gene and comparison with DR5.
Main Results:
- A functional intronic p53 binding site (p53BS) was identified in the DR4 gene.
- p53 directly binds to the DR4 p53BS in cells upon activation.
- The DR4 p53BS mediates p53-dependent transcriptional activation of the DR4 gene.
Conclusions:
- DR4 is a direct transcriptional target of the tumor suppressor p53.
- p53 regulates DR4 expression through a novel intronic p53 binding site.
- This finding elucidates a new mechanism in p53-mediated apoptosis pathways.
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