p53 upregulates death receptor 4 expression through an intronic p53 binding site

Xiangguo Liu1, Ping Yue, Fadlo R Khuri

  • 1Winship Cancer Institute, Emory University School of Medicine, 1365-C Clifton Road, Clinical Building C-3088, Atlanta, GA 30322, USA.

Cancer Research
|August 4, 2004
PubMed

Insights

The tumor suppressor p53 directly regulates Death Receptor 4 (DR4) gene expression. This regulation occurs through a specific binding site within the DR4 gene, confirming DR4 as a p53 target gene.

Area of Science:

  • Molecular Biology
  • Cancer Biology
  • Genetics

Background:

  • Death Receptor 4 (DR4) is a key mediator of apoptosis, activated by TRAIL.
  • Previous research indicated a p53-dependent regulation of DR4 expression.

Purpose of the Study:

  • To determine if DR4 is a direct p53 target gene.
  • To identify the regulatory mechanism of p53 on DR4 expression.

Main Methods:

  • Chromatin immunoprecipitation assays to assess p53 binding to the DR4 gene.
  • Luciferase reporter assays to evaluate transcriptional activity regulated by the p53 binding site.
  • Analysis of p53 binding site (p53BS) in the DR4 gene and comparison with DR5.

Main Results:

  • A functional intronic p53 binding site (p53BS) was identified in the DR4 gene.
  • p53 directly binds to the DR4 p53BS in cells upon activation.
  • The DR4 p53BS mediates p53-dependent transcriptional activation of the DR4 gene.

Conclusions:

  • DR4 is a direct transcriptional target of the tumor suppressor p53.
  • p53 regulates DR4 expression through a novel intronic p53 binding site.
  • This finding elucidates a new mechanism in p53-mediated apoptosis pathways.

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