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[Terson syndrome. A case report].

D Hejcmanová1, E Rencová, Z Chovancová

  • 1Ocní klinika FN, Hradec Králové. hejcmanovad@lfhk.cuni.cz

Ceska a Slovenska Oftalmologie : Casopis Ceske Oftalmologicke Spolecnosti a Slovenske Oftalmologicke Spolecnosti
|September 17, 2004
PubMed
Summary

Severe brain injury can cause Terson syndrome, leading to unilateral retrohyaloid and vitreous hemorrhage. This occurs due to intracranial hypertension affecting optic nerve sheath structures, causing leakage into the vitreous.

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Area of Science:

  • Ophthalmology
  • Neurology
  • Trauma Medicine

Background:

  • Subdural hematomas following severe brain injury can lead to ocular complications.
  • Terson syndrome is characterized by vitreous hemorrhage associated with intracranial events.

Observation:

  • Detailed funduscopy and fluorescein angiography were used to examine patients.
  • A specific site of fluorescein leakage was identified in the lamina limitans interna, extending into the vitreous.

Findings:

  • Fluorescein angiography pinpointed a leakage site at the optic disc margin.
  • The source of hyperfluorescence intensified over time, even after vitreous hemorrhage removal.

Implications:

  • This suggests intracranial hypertension impacts peripapillary tissues via the optic nerve sheath's intervaginal space.

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  • Understanding this mechanism aids in diagnosing and managing Terson syndrome after brain injury.