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Published on: July 2, 2018
[Clinical aspects and haemodynamic parameters for monitoring patients with acute myocardial infarct (AIM)]
Ejub Halilović1, Muharem Merić, Ibro Terzić
1Zavod za fiziologiju, Medicinski fakultet Univerziteta u Tuzli.
Insights
Clinical and hemodynamic assessments are crucial for identifying heart weakness following acute myocardial infarction (AMI). Low heart tones, gallop rhythm, and lung failure are key indicators in AMI patients.
Area of Science:
- Cardiology
- Internal Medicine
Context:
- Acute myocardial infarction (AMI) often leads to localized or diffuse disturbances in myocardial contractility.
- Repeated AMI, particularly with high occlusive coronary disease, can cause significant systolic and diastolic heart dysfunction.
Purpose:
- To emphasize the importance of clinical and hemodynamic parameters in detecting heart failure in patients with AMI.
- To analyze the correlation between clinical signs and hemodynamic measurements in AMI.
Summary:
- This retrospective study analyzed 86 AMI patients (average age 58.42), with 60.46% experiencing front wall and 39.54% back wall infarcts.
- Patients underwent clinical, electrocardiographic, and hemodynamic monitoring using right heart catheterization with a Swan-Ganz catheter.
- Clinical findings revealed silent heart tones (33%), systolic murmurs (19%), gallop rhythm (26%), and lung failure (22%). Silent heart tones, gallop rhythm, and lung failure were the most reliable indicators of heart weakness.
Impact:
- Identifies key clinical indicators (low heart tones, gallop rhythm, lung failure) for early detection of heart weakness in AMI.
- Utilizes advanced hemodynamic monitoring to stratify patients, aiding in targeted management strategies.
- Provides valuable insights for clinicians managing patients post-myocardial infarction.
Abstract:
In acute myocardial infarct (AIM) incomes to a local disturbances in contractility: hypokinesia, akinesia, diskinesia, asynchronia. With repeated AIM esspecially in high oclussive coronary disease incomes to diffuse disturbance in contractility of myocard, with disorder in systolic and diasystolic function of heart. The aim is to point on significance of clinical and haemodynamic parameters in revealing heart weakness in AIM. By retrograded analysis in our study was embraced 86 patients deseased from AIM, average age of 58.42 years. Front wall infarct had 60,46%, backwall infarct had 39.54% of deseased patients. Patients were monitored clinically, electrocardiographically and haemodynamically with microcatetherisation of the right heart by Schwan-Ganz threeluminal thermodilutional catether with regulation of heart minute volumen. By haemodynamic values they were splitted in four groups. In clinical study, silent heart tones had 33%, systolic murmurs on ictus 19%, gallop rhythm 26% and lung failure 22% of patients. The most reliable clinical sign for heart weakness were low heart tones and gallop rhythm as well as lung failure. By haemodynamic values aquired by microcatetherisation of right heart with regulation of minute heart volumen, deseased with AIM were 15% in first group, 28% in second group, 3% in third group and 54% in fourth group of patients.
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