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Creatine supplementation does not decrease total plasma homocysteine in chronic hemodialysis patients
Youri E C Taes1, Joris R Delanghe, Dirk De Bacquer
1Laboratory Clinical Chemistry, University Hospital Ghent, Belgium. youri.taes@Ugent.be
Insights
Creatine supplementation does not lower homocysteine levels in hemodialysis patients already receiving high-dose vitamin therapy. This study found no significant changes in homocysteine (tHcy) concentrations with creatine use.
Area of Science:
- Nephrology
- Biochemistry
- Nutritional Science
Background:
- Hyperhomocysteinemia is common in chronic hemodialysis patients.
- Standard vitamin therapy (folic acid, B12, B6) often fails to normalize homocysteine (tHcy).
- Previous animal studies suggested creatine may lower tHcy in uremia.
Purpose of the Study:
- To investigate the effect of creatine supplementation on plasma homocysteine (tHcy) levels.
- To assess creatine's impact in hemodialysis patients already replete with vitamins.
- To evaluate creatine uptake and its correlation with tHcy.
Main Methods:
- A randomized, placebo-controlled crossover study involving 45 hemodialysis patients.
- Patients received 2 g/day of creatine or placebo for 4 weeks, with a 4-week washout period.
- Measurements included plasma tHcy, creatine, Kt/V(urea), vitamin levels, and inflammatory/nutritional markers.
Main Results:
- All patients presented with elevated tHcy (mean 21.2 +/- 5.6 micromol/L).
- Creatine supplementation increased plasma and red blood cell creatine levels, confirming uptake.
- Creatine did not significantly alter tHcy concentrations or show a correlation with tHcy levels.
Conclusions:
- Daily creatine supplementation (2 g/day) does not reduce tHcy in chronic dialysis patients.
- The findings indicate creatine is ineffective for lowering tHcy in this specific patient population.
- No adverse effects on routine biochemistry, nutritional status, or vitamin levels were observed.
Background:
Hyperhomocysteinemia is present in the majority of chronic hemodialysis patients. Treatment with folic acid, vitamin B12, and vitamin B6 cannot fully normalize plasma homocysteine concentrations (tHcy). Previously we have demonstrated the tHcy-lowering effect of creatine supplementation in an animal model of uremia (Kidney Int 64:1331-1337, 2003). The present study investigates the effects of creatine supplementation on tHcy in a vitamin-repleted chronic hemodialysis population.
Methods:
Forty-five hemodialysis patients receiving folic acid and vitamin B6 and B12 were included. Patients were treated with creatine (2 g/day) or placebo during 2 treatment periods of 4 weeks, separated by a washout of 4 weeks. Plasma tHcy, creatine, Kt/V(urea), folic acid, vitamin B12, and routine biochemistry were determined, as well as the prognostic inflammatory and nutritional index.
Results:
All patients had elevated tHcy concentrations (21.2 +/- 5.6 micromol/L). Creatine treatment resulted in increased plasma and red blood cell creatine levels, documenting uptake of creatine. Creatine did not affect tHcy concentrations. There was no relationship between plasma creatine concentrations and tHcy concentrations. No changes in body weight, routine biochemistry, nutritional status, folic acid, or vitamin B12 were observed during the study.
Conclusion:
Creatine supplementation at a rate of 2 g/day does not further decrease tHcy concentrations in chronic dialysis patients already treated with high dose folic acid, vitamin B6, and B12 supplementation.
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