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Proper folding of the antifungal protein PAF is required for optimal activity
Florentine Marx1, Willibald Salvenmoser, Lydia Kaiserer
1Department of Molecular Biology, Medical University of Innsbruck, Peter-Mayr Strasse 4B/III, 6020 Innsbruck, Austria. florentine.marx@uibk.ac.at
Abstract:
The Penicillium chrysogenumantifungal protein PAF is secreted into the supernatant after elimination of a preprosequence. PAF is actively internalized into the hyphae of sensitive molds and provokes growth retardation as well as changes in morphology. Thus far, no information is available on the exact mode of action of PAF, nor on the function of its prosequence in protein activity. Therefore, we sought to investigate the effects of secreted PAF as well as of intracellularly retained pro-PAF and mature PAF on the sensitive ascomycete Aspergillus nidulans, and transformed this model organism by expression vectors containing 5'-sequentially truncated paf-coding sequences under the control of the inducible P. chrysogenum-derived xylanase promoter. Indirect immunofluorescence staining revealed the localization of recombinant PAF predominantly in the hyphal tips of the transformant Xylpaf1 which expressed prepro-PAF, whereas the protein was found to be distributed intracellularly within all segments of hyphae of the transformants Xylpaf2 and Xylpaf3 which expressed pro-PAF and mature PAF, respectively. Growth retardation of Xylpaf1 and Xylpaf3 hyphae was detected by proliferation assays and by light microscopy analysis. Using transmission electron microscopy of ultrathin hyphal sections a marked alteration of the mitochondrial ultrastructure in Xylpaf1 was observed and an elevated amount of carbonylated proteins pointed to severe oxidative stress in this strain. The effects induced by secreted recombinant PAF resembled those evoked by native PAF. The results give evidence that properly folded PAF is a prerequisite for its activity.
Insights
The Penicillium chrysogenum antifungal protein (PAF) requires proper folding to inhibit fungal growth. Studies show that correctly folded PAF, when secreted, effectively halts mold development and alters cell structure, highlighting its importance in antifungal activity.
Area of Science:
- Mycology
- Protein Biochemistry
- Molecular Biology
Background:
- Penicillium chrysogenum antifungal protein (PAF) is secreted and internalized by sensitive molds, causing growth inhibition and morphological changes.
- The precise mechanism of PAF action and the role of its prosequence remain largely uncharacterized.
Purpose of the Study:
- To investigate the effects of secreted PAF, pro-PAF, and mature PAF on Aspergillus nidulans.
- To elucidate the role of the prosequence and proper folding in PAF's antifungal activity.
Main Methods:
- Genetic transformation of Aspergillus nidulans with truncated paf-coding sequences under an inducible promoter.
- Indirect immunofluorescence microscopy for protein localization.
- Proliferation assays and light microscopy for growth analysis.
- Transmission electron microscopy for ultrastructural examination.
- Measurement of carbonylated proteins to assess oxidative stress.
Main Results:
- Recombinant PAF localization varied based on the expressed form: prepro-PAF concentrated at hyphal tips, while pro-PAF and mature PAF distributed intracellularly.
- Both secreted prepro-PAF and mature PAF induced significant growth retardation in Aspergillus nidulans.
- Ultrastructural analysis revealed mitochondrial alterations and elevated oxidative stress in hyphae expressing prepro-PAF.
- The observed effects of recombinant PAF mimicked those of native PAF.
Conclusions:
- Proper folding of PAF is essential for its antifungal activity.
- Secreted PAF effectively inhibits fungal growth, suggesting its potential as an antifungal agent.
- The prosequence may play a role in protein folding or localization, influencing overall activity.
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