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Related Experiment Videos

Influenza, autoimmunity and atherogenesis.

Victor S Gurevich1

  • 1Mechnicov's State Medical Academy, The Center of Atherosclerosis and Lipid Disorders, Chair of Cardiology, 194291 pr. Kultury 4, CMSD-122, Saint Petersburg, Russia. Gur@cards.lanck.net

Autoimmunity Reviews
|February 22, 2005
PubMed
Summary

Influenza infection may accelerate cardiovascular disease by triggering autoimmune responses. This review explores how viral infections can lead to autoimmune mechanisms that worsen atherosclerosis and increase cardiovascular mortality.

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Autoimmune nature of influenza atherogenicity.

Annals of the New York Academy of Sciences·2005

Area of Science:

  • Cardiovascular Medicine
  • Infectious Diseases
  • Immunology

Background:

  • Influenza epidemics are linked to increased cardiovascular disease (CVD) mortality.
  • Existing data suggest a connection between influenza infection and accelerated coronary atherosclerosis.
  • Autoimmune mechanisms are implicated in the progression of cardiovascular complications following influenza.

Purpose of the Study:

  • To review clinical and experimental data explaining accelerated coronary atherosclerosis in influenza infection.
  • To explore the role of autoimmune mechanisms in influenza-associated cardiovascular disease.
  • To discuss potential cellular and humoral autoimmune pathways involved in atheromatous lesion development.

Main Methods:

  • Review of existing clinical trial data.

Related Experiment Videos

  • Analysis of experimental findings on influenza and atherosclerosis.
  • Examination of proposed autoimmune mechanisms.

    • Main Results:

    • Influenza infection is associated with elevated cardiovascular death rates.
    • Autoimmune responses, both cellular and humoral, are proposed to contribute to atherosclerosis.
    • These autoimmune pathways may drive the onset and progression of atheromatous lesions.

    Conclusions:

    • Influenza infection can accelerate coronary atherosclerosis through autoimmune pathways.
    • Both cellular and humoral immunity play a role in the development of atheromatous lesions.
    • Understanding these mechanisms is crucial for managing cardiovascular risks during influenza.