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Exercise induced arterial hypoxemia in swimmers
S S Spanoudaki1, M D Maridaki, P M Myrianthefs
1Division of Sports Medicine and Biology of Exercise, Laboratory of Functional Anatomy,TEFAA University of Athens, Dafni, Greece.
The Journal of Sports Medicine and Physical Fitness
|March 11, 2005
Summary
Highly trained swimmers may experience mild exercise-induced arterial hypoxemia (EIAH) after intense freestyle swimming. Ex-swimmers did not show this significant drop in arterial oxygen saturation.
Area of Science:
- Sports Medicine
- Cardiopulmonary Physiology
- Exercise Science
Background:
- Exercise-induced arterial hypoxemia (EIAH) is a condition characterized by reduced arterial oxygenation during physical exertion.
- While EIAH is documented in runners and cyclists, its occurrence in swimmers has been less understood.
- Understanding EIAH in swimmers is crucial for optimizing performance and health in aquatic athletes.
Purpose of the Study:
- To investigate the incidence and characteristics of EIAH in highly trained swimmers.
- To compare oxygenation levels in swimmers versus ex-swimmers post-exercise.
- To determine if freestyle swimming at maximal effort induces EIAH.
Main Methods:
- Study involved 8 highly trained male swimmers and 8 male ex-swimmers.
- Subjects performed a 200-meter freestyle swim at maximum effort.
- Hemoglobin oxygen saturation (SaO2%) was measured pre- and immediately post-exercise using a finger pulse oximeter.
Main Results:
- Highly trained swimmers exhibited a statistically significant decrease in SaO2% from 98.3% to 94% (p<0.01) post-exercise.
- Ex-swimmers showed no significant change in SaO2% (98.4% to 96.8%).
- The observed 4% decrease in SaO2% in swimmers was classified as mild EIAH.
Conclusions:
- Highly trained swimmers may develop mild EIAH following maximal effort 200-meter freestyle swimming.
- Ex-swimmers did not demonstrate significant EIAH under the same conditions.
- These findings highlight a potential physiological response to intense swimming in elite athletes.