Related Experiment Video

Updated: Aug 11, 2026

Ascending Aortic Constriction in Rats for Creation of Pressure Overload Cardiac Hypertrophy Model
10:18

Ascending Aortic Constriction in Rats for Creation of Pressure Overload Cardiac Hypertrophy Model

Published on: June 29, 2014

Comparison of cardiac function and valvular damage in children with and without adenotonsillar hypertrophy

Aysenur Pac1, Ahmet Karadag, Hanifi Kurtaran

  • 1Fatih University Faculty of Medicine, Department of Pediatrics and Pediatric Cardiology, Ankara, Turkey.

International Journal of Pediatric Otorhinolaryngology
|March 15, 2005
PubMed

Insights

Children with adenotonsillar hypertrophy showed shortened tricuspid end-diastolic time and potential silent carditis. Further research is needed to confirm the link between enlarged tonsils and cardiac dysfunction in pediatric patients.

Area of Science:

  • Pediatric Cardiology
  • Otorhinolaryngology

Background:

  • Adenotonsillar hypertrophy is common in children.
  • It can lead to various health issues, including potential cardiac complications.

Purpose of the Study:

  • To compare cardiac function in children with and without adenotonsillar hypertrophy.
  • To investigate the potential cardiac effects of adenotonsillar hypertrophy.

Main Methods:

  • 28 children with adenotonsillar hypertrophy and 35 healthy controls were studied.
  • Echocardiographic and electrocardiographic examinations were performed.
  • Statistical analysis included chi-square and t-tests.

Main Results:

  • Shortened tricuspid end-diastolic time was observed in children with adenotonsillar hypertrophy.
  • Five patients with hypertrophy developed cardiac valve damage (silent carditis).
  • No significant difference in right ventricular function was found between groups.

Conclusions:

  • Adenotonsillar hypertrophy may be associated with shortened tricuspid end-diastolic time.
  • Silent carditis is a potential complication in children with adenotonsillar hypertrophy.
  • Further investigation into the link between tonsillar hypertrophy and cardiac dysfunction is warranted.
Abstract

More Related Videos

A Model of Cardiac Remodeling Through Constriction of the Abdominal Aorta in Rats07:31

A Model of Cardiac Remodeling Through Constriction of the Abdominal Aorta in Rats

Published on: December 2, 2016

A Closed-chest Model to Induce Transverse Aortic Constriction in Mice08:10

A Closed-chest Model to Induce Transverse Aortic Constriction in Mice

Published on: April 5, 2018

Related Concept Videos

Mitral Regurgitation I: Introduction01:20

Mitral Regurgitation I: Introduction

Mitral regurgitation is characterized by the backward circulation of blood from the left ventricle to the left atrium during systole, a phase of the cardiac cycle when the heart contracts and pumps blood out of the chambers. This abnormal flow occurs primarily due to the dysfunction of the mitral valve or its supporting structures, which include the mitral leaflets, chordae tendineae, annulus, and papillary muscles.Etiology and Mechanisms:Primary Mitral Regurgitation: This type arises from...
Mitral Stenosis I: Introduction01:22

Mitral Stenosis I: Introduction

Mitral Valve Stenosis (MVS) is a heart condition where the mitral valve narrows, impeding blood circulation from the left atrium to the left ventricle. The etiology and pathophysiology of this condition are multifaceted, leading to a cascade of cardiovascular complications.Causes of Mitral Valve StenosisRheumatic Heart Disease: It is the main cause of mitral valve stenosis, particularly in developing nations. This condition arises from rheumatic fever, an inflammatory illness resulting from...
Mitral Stenosis II: Clinical features and Diagnostic Tests01:23

Mitral Stenosis II: Clinical features and Diagnostic Tests

Mitral stenosis is a heart condition in which the mitral valve, which allows blood to flow from the left atrium to the left ventricle, becomes narrowed or stenotic. This narrowing hinders blood flow and leads to clinical symptoms requiring specific medical evaluations and management strategies. The following overview outlines the clinical symptoms, assessments, diagnostic findings, prevention methods, and treatments for mitral stenosis.Clinical ManifestationsDyspnea (shortness of breath): This...
Aortic Regurgitation II: Clinical Features and Diagnostic Tests01:22

Aortic Regurgitation II: Clinical Features and Diagnostic Tests

Aortic valve regurgitation (AR) occurs when the aortic valve fails to close properly, allowing blood to flow backward from the aorta into the left ventricle. This backflow can result in two distinct clinical presentations: acute and chronic AR, each characterized by its own set of symptoms and physical findings.Acute Aortic RegurgitationAcute AR presents with a sudden onset of severe symptoms. Patients typically experience profound dyspnea (shortness of breath), chest pain, and signs of left...
Rheumatic Heart Disease II: Clinical Manifestations and Diagnostic Studies01:22

Rheumatic Heart Disease II: Clinical Manifestations and Diagnostic Studies

The key clinical manifestations of Rheumatic heart disease (RHD) include several distinct cardiac symptoms.Carditis, a hallmark of acute rheumatic fever, involves inflammation of the heart's endocardium, myocardium, and pericardium. Chronic RHD often results from recurrent episodes of carditis. Its symptoms include the following:Murmurs are caused by valvular damage, especially to the mitral and aortic valves. Mitral stenosis or regurgitation is common, with characteristic heart murmurs...
Cardiomyopathy III: Hypertrophic Cardiomyopathy01:29

Cardiomyopathy III: Hypertrophic Cardiomyopathy

Hypertrophic cardiomyopathy, or HCM, is an autosomal dominant genetic disorder characterized by asymmetric left ventricular hypertrophy without ventricular dilation. It is more common in men and is typically diagnosed in young, athletic adults.EtiologyHCM is primarily genetic and is caused by mutations in genes encoding sarcomeric proteins. Researchers have identified over 1400 mutations across at least 11 different genes. Among these, the most frequently occurring mutations are found in the...