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Conjugal transfer of imipenem resistance in Bacteroides fragilis

K Bandoh1, K Watanabe, Y Muto

  • 1Institute of Anaerobic Bacteriology, Gifu University School of Medicine, Japan.

Insights

Imipenem resistance transfer in Bacteroides fragilis was studied. A 13.6-kb plasmid mediated this transfer, suggesting a conjugation system for imipenem resistance gene spread.

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Imipenem is a critical antibiotic for treating serious infections.
  • Carbapenem resistance, particularly in anaerobic bacteria like Bacteroides fragilis, poses a significant clinical challenge.
  • Understanding resistance mechanisms and transfer is crucial for effective antimicrobial stewardship.

Purpose of the Study:

  • To investigate the mechanism and transfer of imipenem resistance in a clinical isolate of Bacteroides fragilis.
  • To identify the genetic element responsible for imipenem resistance.
  • To characterize the mode of gene transfer.

Main Methods:

  • Filter mating technique for bacterial conjugation experiments.
  • Plasmid DNA isolation and characterization.
  • Electroporation for bacterial transformation.
  • Detection of metallo-beta-lactamase activity.

Main Results:

  • Imipenem resistance was successfully transferred from a clinical isolate (B. fragilis 10-73) to a recipient strain (TM4000) at a frequency of 10(-6).
  • The transfer was mediated by a ~13.6 kb plasmid (pBFUK1) and associated with the production of an imipenem-hydrolyzing metallo-beta-lactamase.
  • Resistance transfer exhibited characteristics of conjugation, being DNase-resistant and not occurring via sterile filtrate.

Conclusions:

  • A novel plasmid-mediated imipenem resistance mechanism involving a metallo-beta-lactamase was identified in Bacteroides fragilis.
  • The findings suggest that conjugation is the primary mechanism for the spread of this imipenem resistance gene within B. fragilis populations.
  • This study highlights the potential for rapid dissemination of antibiotic resistance in clinically important anaerobic bacteria.

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