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[Facial paralysis and chicken-pox].
Revue Neurologique
|January 1, 1992
Summary
A 24-year-old man developed facial palsy after chickenpox, indicating a potential immune response. Despite treatment, some facial muscle weakness persisted, suggesting possible long-term effects.
Area of Science:
- Neurology
- Immunology
- Infectious Diseases
Background:
- Chickenpox (varicella-zoster virus) can lead to neurological complications.
- Facial palsy, a common neurological symptom, can occur post-viral infections.
Observation:
- A 24-year-old male presented with right-sided facial palsy 10 days post-chickenpox.
- Cerebrospinal fluid analysis revealed albumino-cytologic dissociation, an uncommon finding in similar cases.
- The patient exhibited persistent upper facial muscle paresis 2 months after symptom onset.
Findings:
- The presence of albumino-cytologic dissociation in cerebrospinal fluid suggests an immune-mediated mechanism underlying the facial palsy.
- Treatment with prednisone and aciclovir did not result in complete recovery of facial nerve function.
Implications:
- This case highlights a rare immune-mediated complication of chickenpox manifesting as facial palsy.
- The findings suggest that prompt diagnosis and management of such post-infectious neurological complications are crucial.
- Further research may elucidate the specific immunopathogenesis and long-term outcomes of VZV-associated facial palsies.