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[Idiopathic lumbosacral plexopathy].

P Seror1, T Maisonobe, K Viala

  • 1Laboratoire d'Electromyographie, Hôpital de la Salpêtriere, Paris. p.seror@wanadoo.fr

Presse Medicale (Paris, France : 1983)
|August 16, 2005
PubMed
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This summary is machine-generated.

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Lumbosacral plexopathy, similar to Parsonage-Turner syndrome, causes leg pain and weakness. This case highlights slow recovery despite various treatments, emphasizing the need to rule out other causes for this debilitating condition.

Area of Science:

  • Neurology
  • Clinical Neuroscience

Background:

  • Lumbosacral plexopathy is the lower limb equivalent of neuralgic amyotrophy (Parsonage-Turner syndrome).
  • It is recognized in diabetic patients as Bruns-Garland syndrome.

Observation:

  • A 47-year-old woman presented with unilateral leg neuropathy.
  • The condition was neither radicular nor truncal in origin.
  • Improvement was slow and unaffected by administered treatments.

Findings:

  • Lumbosacral plexopathy presents with severe unilateral or bilateral leg pain, motor deficits, and sensory loss.
  • Electrodiagnostic studies reveal significant acute motor and sensory axonal loss.
  • Recovery typically spans 6–36 months, often with incomplete resolution.

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Implications:

  • Accurate diagnosis of lumbosacral plexopathy necessitates excluding other radicular, plexal, and truncal etiologies.
  • Treatment involves a multimodal approach including analgesics, neuropathic pain agents, corticosteroids, and rehabilitation.
  • Severe cases may necessitate long-term immunosuppressive therapy.