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[Ineffective atrial pacing and cardiac arrest in a 80-year old woman with hyperkalemia]
Jacek Lelakowski1, Agnieszka Czunko, Jacek Majewski
1Oddział Kliniczny Elektrokardiologii Krakowskiego Szpitala Specjalistycznego im. Jana Pawła II, Kraków.
Insights
This case study highlights how severe hyperkalemia in an elderly patient on ACE inhibitors and spironolactone caused ineffective atrial pacing and nodal rhythm. Normalizing potassium levels resolved these electrocardiogram changes.
Area of Science:
- Cardiology
- Electrophysiology
- Nephrology
Background:
- Elderly patients on dual therapy with angiotensin-converting enzyme (ACE) inhibitors and spironolactone are at risk for hyperkalemia.
- Pacemaker implantation, particularly AAI mode, can be affected by electrolyte imbalances.
Observation:
- An 80-year-old woman presented with marked hyperkalemia during chronic treatment with ACE inhibitors and spironolactone.
- Electrocardiogram (ECG) revealed a nodal rhythm and ineffective AAI pacing, consistent with hyperkalemia.
Findings:
- Hyperkalemia was the likely cause of the observed ECG abnormalities, including nodal rhythm and ineffective atrial pacing.
- Normalization of serum potassium levels led to the resolution of all ECG changes.
Implications:
- Clinicians should be vigilant for hyperkalemia in patients on ACE inhibitors and spironolactone, especially those with pacemakers.
- Electrolyte disturbances can significantly impact cardiac pacing function and ECG interpretation.
Abstract:
A case of 80-year-old woman with marked hyperkalemia in the course of chronic treatment with angiotensin converting enzyme (ACE) inhibitors and spironolactone is presented. AAI pacemaker was implanted three months ago. The ECG revealed a nodal rhythm, ineffective AAI pacing and other typical features of hyperkalemia. After normalisation of potassium level the ECG changes disappeared. Potential mechanism of the nodal rhythm during ineffective atrial pacing is discussed.
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