Reactive oxygen species

Same author

A GPX1-OSBPL8 axis mediates noncanonical in vivo ferroptosis and cancer growth suppression.

Cell·2026

Same author

Targeting PRDX6-dependent localization and function of GPX4 enhances ferroptosis-mediated tumor suppression.

Molecular cell·2025

Same author

Endothelial CYB5R1 is a Coenzyme Q reductase that suppresses ferroptosis and atherosclerosis.

Research square·2025

Same author

Targeting LxCxE Cleft Pocket of Retinoblastoma Protein in Immunosuppressive Macrophages Inhibits Ovarian Cancer Progression.

Cancer immunology research·2025

Same author

Antiferroptotic Drugs Reduce sFlt-1 Release and Placenta Damage in Preeclampsia.

Hypertension (Dallas, Tex. : 1979)·2025

Same author

Author Correction: Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis.

Nature cell biology·2025

Same journal

The authors reply.

Critical care medicine·2026

Same journal

Attracting Emergency Medicine Graduates to Surgical Critical Care Training Programs.

Critical care medicine·2026

Same journal

The authors reply.

Critical care medicine·2026

Same journal

Beyond a Snapshot: Tracking Family Prognostic Expectations in the ICU.

Critical care medicine·2026

Same journal

The authors reply.

Critical care medicine·2026

Same journal

Plasma Levels of Soluble ST2 Reflect Extrapulmonary Organ Dysfunction and Predict Outcomes in Acute Respiratory Failure: Beware of Potential Confounders.

Critical care medicine·2026

Related Experiment Videos

Reactive oxygen species

Related Concept Videos

A GPX1-OSBPL8 axis mediates noncanonical in vivo ferroptosis and cancer growth suppression.

Targeting PRDX6-dependent localization and function of GPX4 enhances ferroptosis-mediated tumor suppression.

Endothelial CYB5R1 is a Coenzyme Q reductase that suppresses ferroptosis and atherosclerosis.

Targeting LxCxE Cleft Pocket of Retinoblastoma Protein in Immunosuppressive Macrophages Inhibits Ovarian Cancer Progression.

Antiferroptotic Drugs Reduce sFlt-1 Release and Placenta Damage in Preeclampsia.

Author Correction: Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis.

The authors reply.

Attracting Emergency Medicine Graduates to Surgical Critical Care Training Programs.

The authors reply.

Beyond a Snapshot: Tracking Family Prognostic Expectations in the ICU.

The authors reply.

Plasma Levels of Soluble ST2 Reflect Extrapulmonary Organ Dysfunction and Predict Outcomes in Acute Respiratory Failure: Beware of Potential Confounders.

Related Experiment Videos

Reactive oxygen species

Related Concept Videos

Related Articles

A GPX1-OSBPL8 axis mediates noncanonical in vivo ferroptosis and cancer growth suppression.

Targeting PRDX6-dependent localization and function of GPX4 enhances ferroptosis-mediated tumor suppression.

Endothelial CYB5R1 is a Coenzyme Q reductase that suppresses ferroptosis and atherosclerosis.

Targeting LxCxE Cleft Pocket of Retinoblastoma Protein in Immunosuppressive Macrophages Inhibits Ovarian Cancer Progression.

Antiferroptotic Drugs Reduce sFlt-1 Release and Placenta Damage in Preeclampsia.

Author Correction: Endoplasmic reticulum-mitochondria contacts are prime hotspots of phospholipid peroxidation driving ferroptosis.

The authors reply.

Attracting Emergency Medicine Graduates to Surgical Critical Care Training Programs.

The authors reply.

Beyond a Snapshot: Tracking Family Prognostic Expectations in the ICU.

The authors reply.

Plasma Levels of Soluble ST2 Reflect Extrapulmonary Organ Dysfunction and Predict Outcomes in Acute Respiratory Failure: Beware of Potential Confounders.