Related Experiment Videos

Toward a unified hypothesis of interneuronal modulation

Daniel V Madison¹, A Rory McQuiston

¹Department of Molecular and Cellular Physiology, Stanford University School of Medicine, CA 94305-5345, USA. madison@stanford.edu

The Journal of Physiology

|December 24, 2005

Summary

No abstract available in PubMed .

Related Concept Videos

Articles linked to this work by shared authors, journal, and citation graph.

Acute fentanyl and xylazine co-exposure uniquely increase the excitability of dopamine type 2 receptor-expressing striatal medium spiny neurons.

Journal of neurophysiology·2025

Challenges and lessons learned in attempts to develop in vitro models for ultrasound neuromodulation using local field potential measurements in hippocampal tissue.

Biophysical journal·2025

Sustained fentanyl exposure inhibits neuronal activity in dissociated striatal neuronal-glial cocultures through actions independent of opioid receptors.

Journal of neurophysiology·2024

Progressive Degeneration and Adaptive Excitability in Dopamine D1 and D2 Receptor-Expressing Striatal Neurons Exposed to HIV-1 Tat and Morphine.

Cellular and molecular neurobiology·2022

Chronic Morphine Induces IL-18 in Ileum Myenteric Plexus Neurons Through Mu-opioid Receptor Activation in Cholinergic and VIPergic Neurons.

Journal of neuroimmune pharmacology : the official journal of the Society on NeuroImmune Pharmacology·2022

Chloride channels with ClC-1-like properties differentially regulate the excitability of dopamine receptor D1- and D2-expressing striatal medium spiny neurons.

American journal of physiology. Cell physiology·2022

See all related articles