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Fibrocartilaginous emboli.

¹Department of Veterinary Clinical Sciences, Louisiana State University School of Veterinary Medicine, Baton Rouge.

The Veterinary Clinics of North America. Small Animal Practice

|July 1, 1992

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Fibrocartilaginous embolism (FCE) causes acute spinal cord infarction in large dogs. Early diagnosis and recognition of motor neuron deficits are key to prognosis, with most recovery occurring within 10 days.

Area of Science:

Veterinary Neurology
Canine Spinal Cord Diseases

Background:

Fibrocartilaginous embolism (FCE) is a common cause of acute ischemic myelopathy in dogs.
It typically affects large or giant breeds, leading to spinal cord infarction.

Purpose of the Study:

To describe the clinical presentation and diagnostic considerations for FCE.
To discuss the prognostic indicators associated with FCE in canine patients.

Main Methods:

Review of clinical signs suggestive of spinal cord infarction.
Differential diagnosis process to exclude other causes of acute myelopathy.
Evaluation of prognostic factors based on neurological deficits.

Main Results:

Lateralizing and asymmetric neurologic deficits strongly suggest spinal cord infarction.

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Diagnosis relies on excluding other acute compressive myelopathies like trauma or disc herniation.

Lower motor neuron deficits indicate a more guarded prognosis compared to upper motor neuron deficits.

Conclusions:

Fibrocartilaginous embolism is a significant cause of acute ischemic myelopathy in dogs.
Prognosis is influenced by the type of motor neuron deficits observed.
Clinical improvement, if it occurs, is typically seen within the first 10 days of onset.