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Nucleotide sequences specific for Tn1545-like conjugative transposons in pneumococci and staphylococci resistant to

C Poyart-Salmeron1, P Trieu-Cuot, C Carlier

  • 1Unité des Agents Antibactériens, Institut Pasteur, Paris, France.

Insights

Tetracycline resistance in Streptococcus pneumoniae is linked to the tet(M) gene and often involves mobile genetic elements like Tn1545-like transposons. This genetic element, int-Tn, was frequently found in resistant S. pneumoniae but rarely in Staphylococcus species with tet(M).

Area of Science:

  • Microbiology
  • Molecular Biology
  • Genetics

Background:

  • Tetracycline resistance is a growing public health concern.
  • The tet(M) gene is a common determinant of tetracycline resistance.
  • Conjugative transposons, such as Tn1545, facilitate the spread of antibiotic resistance genes.

Purpose of the Study:

  • To investigate the distribution of the tet(M) gene and Tn1545-like conjugative transposons in tetracycline-resistant Streptococcus pneumoniae.
  • To compare the genetic context of tet(M) in S. pneumoniae with that in Staphylococcus species.

Main Methods:

  • Hybridization techniques were used to detect the presence of tet(M) and int-Tn.
  • The study analyzed 47 clinical isolates of Streptococcus pneumoniae and 37 strains of Staphylococcus spp. resistant to tetracycline.

Main Results:

  • All tetracycline-resistant S. pneumoniae isolates possessed the tet(M) gene, which was also associated with minocycline resistance.
  • The int-Tn gene, essential for Tn1545-like transposon mobility, was found in all but one S. pneumoniae isolate.
  • In contrast, int-Tn was detected in only 2 out of 37 tetracycline-resistant Staphylococcus isolates carrying tet(M).

Conclusions:

  • The tet(M) gene is the primary cause of tetracycline and minocycline resistance in the studied S. pneumoniae.
  • Tn1545-like transposons appear to be widely distributed and efficiently mobilized in S. pneumoniae clinical isolates.
  • The genetic environment and mobility of tet(M) differ significantly between S. pneumoniae and Staphylococcus species.

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