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Related Experiment Videos

[Diffuse idiopathic skeletal hyperostosis (D.I.S.H.)].

M Colina1, M Govoni, F De Leonardis

  • 1Sezione di Reumatologia, Dipartimento Medicina Clinica e Sperimentale, Università degli Studi di Ferrara. teocolina@libero.it

Reumatismo
|July 11, 2006
PubMed
Summary
This summary is machine-generated.

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Diffuse idiopathic skeletal hyperostosis (DISH) causes bone overgrowth, particularly in the spine and where tendons attach to bone. This review explores its causes and clinical features.

Area of Science:

  • Rheumatology
  • Orthopedics
  • Metabolic Disorders

Context:

  • Diffuse idiopathic skeletal hyperostosis (DISH) is a common skeletal disorder of unknown origin.
  • It is characterized by excessive bone formation along the spine and at tendon/ligament attachment sites (entheses).
  • DISH frequently co-occurs with metabolic conditions like type 2 diabetes, hypertension, and cardiovascular issues.

Purpose:

  • To review the etiopathogenesis and clinical manifestations of DISH.
  • To differentiate DISH from osteoarthritis, highlighting key pathological differences.
  • To discuss the spinal and extraspinal skeletal involvement in DISH.

Summary:

  • DISH involves hyperostosis of the anterolateral spine, potentially causing ankylosis, and ossification of extraspinal entheses.

Related Experiment Videos

  • Enthesopathy in DISH affects the anterior longitudinal ligament, forming flowing osteophytes while preserving disc spaces.
  • Appendicular skeleton involvement, especially in the feet, elbows, and patella, is common and can lead to significant disability.

    • Impact:

    • Understanding DISH pathogenesis and clinical presentation is crucial for accurate diagnosis and management.
    • Distinguishing DISH from osteoarthritis is important for appropriate treatment strategies.
    • Recognizing the association with metabolic derangements and potential for severe disability aids in comprehensive patient care.