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Updated: Aug 6, 2026

Primed Mycobacterial Uveitis (PMU) as a Model for Post-Infectious Uveitis
Published on: December 17, 2021
[Etiopathogeny of intraocular pressure modifications in uveitis]
K Souissi1, M A El Afrit, S Trojet
1Service d'Ophtalmologie, Centre Hospitalo-Universitaire Habib Thameur, Tunis, Tunisie. souissikhal@yahoo.fr
Abstract:
Uveitic glaucoma is a secondary form of glaucoma. Treatment is essentially medical, based on antiglaucomatous medications and immunomodulatory therapy. Therefore, some patients may need filtrating surgery to control intraocular pressure. Understanding the cellular and biochemical modifications of aqueous humor that occur during intraocular inflammation and identification of anatomical modifications of the iridocorneal angle, trabecula, pupil, and ciliary body allow physicians to adapt management depending on the different clinical patterns of uveitic glaucoma. We propose a general review of the role of inflammatory mediators and etiopathogenic mechanisms involved in uveitic glaucoma.
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