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Outer membrane proteins of E. coli in the host-pathogen interaction in urinary tract infection

J A Robledo1, A Serrano, G J Domingue

  • 1Department of Urology, Tulane University School of Medicine, New Orleans, Louisiana 70112.

The Journal of Urology
|February 1, 1990
PubMed

Insights

Host factors like low iron and high osmolality in the urinary tract alter Escherichia coli outer membrane protein (Omp) expression, influencing bacterial survival and colonization. This impacts the host-parasite interaction in urinary tract infections.

Area of Science:

  • Microbiology
  • Bacterial Pathogenesis
  • Host-Pathogen Interactions

Background:

  • Escherichia coli (E. coli) outer membrane protein (Omp) expression may be influenced by in vivo growth conditions.
  • Understanding these changes is crucial for deciphering E. coli's ability to colonize the urinary tract.

Purpose of the Study:

  • To investigate the impact of host-derived factors on E. coli outer membrane protein patterns directly from urine.
  • To compare Omp expression in vivo versus in vitro conditions.

Main Methods:

  • Polyacrylamide gel electrophoresis (SDS-PAGE) was used to analyze Omp patterns of E. coli from patient urine.
  • Comparative growth experiments were performed on artificial culture media (ACM) and pooled human urine.
  • In vitro studies manipulated osmolality to assess its effect on porin expression.

Main Results:

  • Two distinct Omp patterns were observed in urinary E. coli strains, with variations in Omp A and a potential Omp A fragment.
  • High molecular weight proteins were linked to iron restriction.
  • Porin expression decreased in urine compared to ACM, correlating with osmolality differences.

Conclusions:

  • Host factors, specifically low iron availability and high osmolality in the urinary tract, significantly influence E. coli surface protein expression.
  • These altered protein profiles may enhance E. coli survival and colonization in the urinary tract.
  • Immune responses targeting porins could play a role in the host-parasite interaction during urinary tract infections.

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