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Related Concept Videos

Hemorrhagic Stroke ll: Pathophysiology01:29

Hemorrhagic Stroke ll: Pathophysiology

A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
Hemorrhagic Stroke l: Introduction01:17

Hemorrhagic Stroke l: Introduction

A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...
Esophageal Varices-II: Clinical Features and Management01:28

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Esophageal varices often manifest as gastrointestinal bleeding episodes, presenting symptoms like hematemesis (vomiting of blood), hematochezia (passing fresh blood via the rectum), and melena (black, tarry stools). Other signs can include weight loss, anorexia, abdominal discomfort, jaundice, pruritus, altered mental status, and muscle cramps.
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Esophageal Varices-I: Introduction01:24

Esophageal Varices-I: Introduction

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Disorders of Hemostasis01:24

Disorders of Hemostasis

Hemostasis, the process that stops bleeding after a blood vessel injury, is crucial for maintaining the integrity of the circulatory system. However, disorders of hemostasis can disrupt this delicate balance, leading to either excessive clotting or bleeding. These disorders can be broadly classified into thromboembolic disorders and bleeding disorders.
Thromboembolic Disorders
Two factors primarily cause thromboembolic conditions.
Ischemic Stroke ll: Pathophysiology01:15

Ischemic Stroke ll: Pathophysiology

An ischemic stroke occurs when a cerebral blood vessel becomes obstructed, most often by a thrombus or embolus, interrupting the delivery of oxygen and glucose to brain tissue. Because neurons rely on continuous aerobic metabolism, energy failure begins within minutes of reduced perfusion. The region receiving the least blood flow becomes the infarct core, an area of irreversible cellular death. Surrounding this core lies the penumbra, a zone of hypoperfused but still viable tissue that is...

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Related Experiment Video

Updated: Jul 20, 2026

The Rabbit Blood-shunt Model for the Study of Acute and Late Sequelae of Subarachnoid Hemorrhage: Technical Aspects
09:00

The Rabbit Blood-shunt Model for the Study of Acute and Late Sequelae of Subarachnoid Hemorrhage: Technical Aspects

Published on: October 2, 2014

[Benckiser's hemorrhage: severe and inevitable?].

C Régis1, N-D Mubiayi, L Devisme

  • 1Clinique d'Obstétrique, Hôpital Jeanne de Flandre, CHRU de Lille, 1, rue Eugène-Avinée, 59037 Lille Cedex. regis.claudia@wanadoo.fr

Journal De Gynecologie, Obstetrique Et Biologie De La Reproduction
|August 31, 2006
PubMed
Summary

Benckiser's hemorrhage, a rare condition from velamentous and vasa previa vessel rupture, has high neonatal mortality. Management is challenging, even with prompt intervention, highlighting the need for better diagnostic strategies.

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Last Updated: Jul 20, 2026

The Rabbit Blood-shunt Model for the Study of Acute and Late Sequelae of Subarachnoid Hemorrhage: Technical Aspects
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Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage
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Double Direct Injection of Blood into the Cisterna Magna as a Model of Subarachnoid Hemorrhage

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Area of Science:

  • Obstetrics and Gynecology
  • Neonatal Medicine
  • Fetal Medicine

Background:

  • Benckiser's hemorrhage is a rare but severe obstetric emergency.
  • It results from the rupture of umbilical cord vessels (velamentous insertion) or placental vessels near the cervix (vasa previa).
  • This condition is associated with extremely high neonatal mortality rates, ranging from 75% to 100%.

Observation:

  • This report details two cases of Benckiser's hemorrhage.
  • One case resulted in neonatal death despite immediate and intensive management.
  • These cases underscore the significant difficulties in managing this obstetric and neonatal emergency.

Findings:

  • Identified risk factors for velamentous cord insertion and vasa previa include multiple pregnancies, low-lying placentas, and specific placental anomalies (bilobed, succenturiate-lobed).
  • The pathophysiological implications of this severe hemorrhage are discussed.
  • Diagnostic methods for fetal bleeding and vasa previa are reviewed.

Implications:

  • Improved understanding of pathophysiology and diagnostic tools may enable the development of sonographic screening strategies for vasa previa.
  • Further research is needed to determine the benefits, risks, limitations, and costs associated with implementing such screening protocols.
  • Effective management strategies for Benckiser's hemorrhage require further investigation and refinement.