Macrophage vitronectin receptor, CD36, and thrombospondin cooperate in recognition of neutrophils undergoing programmed cell death

Phosphatidylethanolamine is progressively exposed in RBCs during storage.

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Factor-induced differentiation and activation of macrophages.

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Downregulation of Mcl-1 has anti-inflammatory pro-resolution effects and enhances bacterial clearance from the lung.

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Combined dysfunctions of immune cells predict nosocomial infection in critically ill patients.

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Cyclin-dependent kinases 7 and 9 specifically regulate neutrophil transcription and their inhibition drives apoptosis to promote resolution of inflammation.

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Macrophage vitronectin receptor, CD36, and thrombospondin cooperate in recognition of neutrophils undergoing

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Macrophage vitronectin receptor, CD36, and thrombospondin cooperate in recognition of neutrophils undergoing