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Identification of autophosphorylation sites of HER2/neu

R Hazan1, B Margolis, M Dombalagian

  • 1Rorer Biotechnology, Inc., King of Prussia, Pennsylvania 19406.

Cell Growth & Differentiation : the Molecular Biology Journal of the American Association for Cancer Research
|January 1, 1990
PubMed

Insights

Researchers identified key autophosphorylation sites (Y1023, Y1248) on the HER2 receptor, crucial for its role in cancers like breast and ovarian malignancies. Additional potential sites (Y1139, Y1222) were also suggested.

Area of Science:

  • Oncology
  • Molecular Biology
  • Biochemistry

Background:

  • HER2 (Human Epidermal growth factor Receptor 2), also known as c-erbB-2, is a growth factor receptor.
  • It shares sequence homology with the epidermal growth factor receptor and is the human homologue of the rat protooncogene neu.
  • HER2 plays a significant role in human malignancies, particularly breast and ovarian cancers.

Purpose of the Study:

  • To identify the specific autophosphorylation sites on the HER2 protein.
  • To further elucidate the molecular mechanisms of HER2 signaling in cancer.

Main Methods:

  • Immunopurification of HER2 protein.
  • Induction of HER2 autophosphorylation.
  • Generation and purification of tryptic phosphopeptides using high-performance liquid chromatography (HPLC).
  • Microsequencing of purified phosphopeptides.

Main Results:

  • Two definitive autophosphorylation sites were identified at tyrosine residues Y1023 and Y1248.
  • Two additional tyrosine phosphopeptides were sequenced, suggesting Y1139 and Y1222 as potential autophosphorylation sites.
  • These findings indicate that HER2 autophosphorylation sites are located in the carboxyl terminus, similar to the epidermal growth factor receptor.

Conclusions:

  • The study unequivocally identified Y1023 and Y1248 as HER2 autophosphorylation sites.
  • Y1139 and Y1222 are proposed as additional autophosphorylation sites, contributing to HER2's function in cancer.
  • Understanding these sites is critical for targeted cancer therapies.

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