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Assessing Functional Recovery of Eupneic Diaphragm Activity Following Unilateral Cervical Spinal Cord Hemisection in Rats
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Changes in expiratory muscle function following spinal cord section.

Krzysztof E Kowalski1, Jaroslaw R Romaniuk, Anthony F DiMarco

  • 1Dept. of Physiology and Biophysics, Case Western Reserve Univ., MetroHealth Medical Center, Rammelkamp Center for Education & Research, 2500 MetroHealth Dr., Cleveland, OH 44109-1998. afd3@cwru.edu).

Journal of Applied Physiology (Bethesda, Md. : 1985)
|December 13, 2006
PubMed
Summary

Spinal cord injury causes significant disuse atrophy in expiratory muscles, leading to reduced airway pressure-generating capacity. This study in cats shows muscle mass loss and fiber type changes after spinalization.

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Area of Science:

  • Physiology
  • Neurology
  • Muscle Biology

Background:

  • Spinal cord injury (SCI) frequently results in disuse atrophy of muscles below the injury level.
  • Expiratory muscles are crucial for respiration and can be affected by SCI.

Purpose of the Study:

  • To investigate the physiological changes in expiratory muscles following spinal cord injury in a feline model.
  • To assess the impact of SCI on muscle mass, fiber type, and pressure-generating capacity of expiratory muscles.

Main Methods:

  • Five cats underwent T(6) spinalization, with physiological parameters assessed before and 6 months post-injury.
  • Parameters measured included muscle fiber typing, cross-sectional area, muscle weight, and airway pressure-generating capacity.
  • A control group of five acute animals was used for comparison.

Main Results:

  • Spinalized cats showed a significant decrease in airway pressure-generating capacity (from 41 to 28 cm H2O).
  • Significant reductions in muscle weight and cross-sectional area were observed in key expiratory muscles (external oblique, internal oblique, transversus abdominis, internal intercostal).
  • A notable increase in fast muscle fiber population occurred in the expiratory muscles of spinalized animals.

Conclusions:

  • Spinalization leads to significant atrophy and fiber-type transformation in expiratory muscles.
  • Reduced muscle mass is the primary cause of the diminished pressure-generating capacity of expiratory muscles after spinal cord injury.