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Preclinical selection of a novel poly(ADP-ribose) polymerase inhibitor for clinical trial
Huw D Thomas1, Christopher R Calabrese, Michael A Batey
1Newcastle University, Northern Institute for Cancer Research, Medical School, Framlington Place, Newcastle upon Tyne NE2 4HH, United Kingdom.
Abstract:
Poly(ADP-ribose) polymerase (PARP)-1 (EC 2.4.2.30) is a nuclear enzyme that promotes the base excision repair of DNA breaks. Inhibition of PARP-1 enhances the efficacy of DNA alkylating agents, topoisomerase I poisons, and ionizing radiation. Our aim was to identify a PARP inhibitor for clinical trial from a panel of 42 potent PARP inhibitors (K(i), 1.4-15.1 nmol/L) based on the quinazolinone, benzimidazole, tricyclic benzimidazole, tricyclic indole, and tricyclic indole-1-one core structures. We evaluated chemosensitization of temozolomide and topotecan using LoVo and SW620 human colorectal cells; in vitro radiosensitization was measured using LoVo cells, and the enhancement of antitumor activity of temozolomide was evaluated in mice bearing SW620 xenografts. Excellent chemopotentiation and radiopotentiation were observed in vitro, with 17 of the compounds causing a greater temozolomide and topotecan sensitization than the benchmark inhibitor AG14361 and 10 compounds were more potent radiosensitizers than AG14361. In tumor-bearing mice, none of the compounds were toxic when given alone, and the antitumor activity of the PARP inhibitor-temozolomide combinations was unrelated to toxicity. Compounds that were more potent chemosensitizers in vivo than AG14361 were also more potent in vitro, validating in vitro assays as a prescreen. These studies have identified a compound, AG14447, as a PARP inhibitor with outstanding in vivo chemosensitization potency at tolerable doses, which is at least 10 times more potent than the initial lead, AG14361. The phosphate salt of AG14447 (AG014699), which has improved aqueous solubility, has been selected for clinical trial.
Insights
Poly(ADP-ribose) polymerase (PARP)-1 inhibitors enhance cancer treatments. Researchers identified AG14447, a potent PARP inhibitor, for clinical trials due to its strong ability to sensitize tumors to chemotherapy and radiation.
Area of Science:
- Biochemistry
- Molecular Biology
- Pharmacology
Background:
- Poly(ADP-ribose) polymerase (PARP)-1 is a key enzyme in DNA repair.
- Inhibiting PARP-1 can potentiate the effects of DNA-damaging cancer therapies.
- Identifying novel PARP inhibitors is crucial for improving cancer treatment efficacy.
Purpose of the Study:
- To identify a potent Poly(ADP-ribose) polymerase (PARP)-1 inhibitor for clinical development.
- To evaluate the chemosensitization and radiosensitization potential of novel PARP inhibitors.
- To assess the in vivo antitumor activity and toxicity of selected PARP inhibitors in combination with chemotherapy.
Main Methods:
- Screened 42 novel Poly(ADP-ribose) polymerase (PARP)-1 inhibitors with diverse core structures.
- Assessed in vitro chemosensitization with temozolomide and topotecan in colorectal cancer cells.
- Measured in vitro radiosensitization and in vivo antitumor activity in mouse xenograft models.
Main Results:
- Several compounds demonstrated superior in vitro chemosensitization and radiosensitization compared to the benchmark AG14361.
- The novel PARP inhibitor AG14447 showed outstanding in vivo chemosensitization potency at well-tolerated doses.
- In vivo efficacy of combinations was not linked to observed toxicity, validating in vitro prescreening.
Conclusions:
- AG14447 is a highly potent Poly(ADP-ribose) polymerase (PARP)-1 inhibitor with significant in vivo chemosensitization capabilities.
- The phosphate salt of AG14447 (AG014699) has been selected for clinical trials due to its enhanced solubility and potency.
- This study validates in vitro assays for prescreening PARP inhibitors and identifies a promising candidate for cancer therapy enhancement.

