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Related Concept Videos

Cardiomyopathy II: Dilated Cardiomyopathy01:30

Cardiomyopathy II: Dilated Cardiomyopathy

Dilated cardiomyopathy, or DCM, is a progressive myocardial disorder characterized by ventricular chamber dilation and contractile dysfunction.EtiologyVarious factors can cause DCM, including hypertension and heavy alcohol intake, which contribute to the weakening and enlargement of the heart muscle. Viral infections, such as Coxsackievirus B, adenoviruses, and influenza, can lead to DCM by causing inflammation and damage to heart tissue. Certain chemotherapeutic agents, including daunorubicin,...
Cardiomyopathy I: Introduction and Classification01:25

Cardiomyopathy I: Introduction and Classification

Cardiomyopathy, or CMP, is a group of diseases affecting the myocardial structure, impairing its ability to pump blood effectively. This condition can lead to arrhythmias, heart failure, or sudden cardiac death.Cardiomyopathies are classified into primary and secondary categories:Primary Cardiomyopathy refers to conditions involving only the heart muscle that are often idiopathic (of unknown cause) or genetic. They primarily affect the myocardium without the involvement of other systemic...
Cardiomyopathy IV: Restrictive Cardiomyopathy01:29

Cardiomyopathy IV: Restrictive Cardiomyopathy

Restrictive cardiomyopathy (RCM) is a rare heart muscle disease characterized by impaired ventricular filling due to stiffened ventricular walls, leading to significant diastolic dysfunction.EtiologyRestrictive cardiomyopathy can arise from both inherited and acquired diseases, many of which are systemic. It is categorized into four main types: infiltrative, storage, non-infiltrative, and endomyocardial diseases.Infiltrative diseases, such as amyloidosis, lead to RCM by depositing amyloid...
Cardiomyopathy V: Interprofessional Care01:29

Cardiomyopathy V: Interprofessional Care

Managing cardiomyopathy involves addressing underlying or precipitating causes, treating heart failure with medications, and implementing dietary changes and a balanced exercise and rest regimen.Lifestyle ModificationsCardiomyopathy patients should adopt a low-sodium diet to reduce fluid retention and manage heart failure. A personalized exercise and rest plan helps maintain physical fitness without overstraining the heart. Avoiding alcohol and tobacco is essential to prevent further damage to...
Mitral Stenosis II: Clinical features and Diagnostic Tests01:23

Mitral Stenosis II: Clinical features and Diagnostic Tests

Mitral stenosis is a heart condition in which the mitral valve, which allows blood to flow from the left atrium to the left ventricle, becomes narrowed or stenotic. This narrowing hinders blood flow and leads to clinical symptoms requiring specific medical evaluations and management strategies. The following overview outlines the clinical symptoms, assessments, diagnostic findings, prevention methods, and treatments for mitral stenosis.Clinical ManifestationsDyspnea (shortness of breath): This...
Cardiomyopathy III: Hypertrophic Cardiomyopathy01:29

Cardiomyopathy III: Hypertrophic Cardiomyopathy

Hypertrophic cardiomyopathy, or HCM, is an autosomal dominant genetic disorder characterized by asymmetric left ventricular hypertrophy without ventricular dilation. It is more common in men and is typically diagnosed in young, athletic adults.EtiologyHCM is primarily genetic and is caused by mutations in genes encoding sarcomeric proteins. Researchers have identified over 1400 mutations across at least 11 different genes. Among these, the most frequently occurring mutations are found in the...

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Related Experiment Video

Updated: Jul 15, 2026

Generation and Characterization of Right Ventricular Myocardial Infarction Induced by Permanent Ligation of the Right Coronary Artery in Mice
10:18

Generation and Characterization of Right Ventricular Myocardial Infarction Induced by Permanent Ligation of the Right Coronary Artery in Mice

Published on: February 1, 2022

Isolated right ventricular dysfunction without myocardial infarction.

Yoritaka Otsuka, Atsushi Kawamura, Shunichi Miyazaki

    International Journal of Cardiology
    |April 17, 2007
    PubMed
    Summary

    This case report details a patient with unstable angina who experienced temporary isolated right ventricular (RV) dysfunction after a blocked right coronary artery was treated. The RV function and hemodynamics improved quickly following percutaneous coronary intervention.

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    Published on: February 1, 2022

    Morphological and Functional Assessment of the Right Ventricle Using 3D Echocardiography
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    Point-of-Care Ultrasound for Peripheral Veno-Arterial Extracorporeal Membrane Oxygenation Without Left Ventricular Venting

    Published on: January 17, 2025

    Area of Science:

    • Cardiology
    • Cardiovascular Imaging
    • Interventional Cardiology

    Background:

    • Right ventricular (RV) dysfunction often complicates acute inferior myocardial infarction.
    • Isolated RV dysfunction without left ventricular involvement is less common.

    Observation:

    • A patient with unstable angina presented with temporary isolated RV dysfunction.
    • The patient had a total occlusion of the proximal right coronary artery (RCA) with collateral circulation from the left coronary artery.
    • Percutaneous coronary intervention (PCI) successfully recanalized the occluded RCA.

    Findings:

    • Echocardiography revealed RV dilatation and severe free wall hypokinesis, with no left ventricular abnormalities.
    • Hemodynamic assessment post-PCI showed elevated right atrial pressure, a non-compliant pattern, and low cardiac output.
    • Cardiac enzymes remained un-elevated, and SPECT imaging confirmed RV free wall uptake of 99mTc-pyrophosphate without left ventricular abnormalities or perfusion defects.

    Implications:

    • This case highlights a unique presentation of temporary isolated RV dysfunction following PCI for RCA occlusion.
    • It underscores the importance of comprehensive echocardiographic and hemodynamic assessment in managing RV dysfunction.
    • The findings suggest that RV dysfunction in this context may be transient and responsive to revascularization, even without elevated cardiac enzymes.