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Cardiac tamponade as complication of active-fixation atrial lead perforations: proposed mechanism and management
Vitaly Geyfman1, Randle H Storm, Serrie C Lico
1Department of Cardiology, Geisinger Medical Center, Danville, Pennsylvania, USA. vgeyfman1@geisinger.edu
Insights
Cardiac tamponade after pacemaker implantation is rare but serious. A conservative management algorithm involving pericardiocentesis and lead repositioning is proposed as safe and effective.
Area of Science:
- Cardiology
- Cardiac Electrophysiology
Background:
- Cardiac tamponade is a rare but serious complication following dual-chamber pacemaker or defibrillator implantation.
- The exact pathophysiology and optimal management strategies for this condition remain incompletely understood.
Observation:
- Three cases of cardiac tamponade after transvenous dual-chamber pacemaker implantation with active-fixation atrial leads were analyzed.
- The common etiology identified was bleeding into the pericardial space due to atrial lead helix perforation through the atrial wall.
Findings:
- Two patients required open thoracotomy for repair of the atrial perforation.
- One patient successfully underwent lead repositioning under fluoroscopic guidance in the electrophysiology laboratory.
Implications:
- A conservative management algorithm is recommended for post-implant cardiac tamponade.
- Percutaneous pericardiocentesis with drainage, followed by fluoroscopically guided lead repositioning with surgical backup, appears safe and effective.
Background:
Cardiac tamponade is a rare complication after implantation of dual chamber pacemaker or defibrillator systems. Its pathophysiology and optimal management are not currently well established.
Methods:
Three cases of cardiac tamponade following successful implantation of transvenous dual chamber pacemakers with active-fixation atrial leads were identified.
Results:
All three patients with post-implant cardiac tamponade were suspected to have the same etiology of bleeding into the pericardial space. This was due to protrusion of the helix of the active-fixation atrial pacing lead through the atrial wall with subsequent abrasion of visceral pericardial layer and bleeding from the atrium through the perforation. In two patients, the perforation sites were visualized and repaired during open thoracotomy in the operating room. The third patient underwent lead repositioning under fluoroscopic guidance in the electrophysiology laboratory.
Conclusion:
Based on the reviewed cases, we describe the pathophysiology of, and recommend a safe conservative algorithm for, the management of cardiac tamponade after successful transvenous lead implantation. Percutaneous pericardiocentesis with placement of the pericardial drain followed by lead repositioning under fluoroscopic guidance with surgical backup appears to be safe and effective.
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