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Related Experiment Videos

Trazodone and ejaculatory inhibition.

Kenneth R Kaufman1, Humbarto Marin, Matthew Menza

  • 1Department of Psychiatry, UMDNJ-Robert Wood Johnson Medical School, New Brunswick, NJ 08901, USA. kaufmakr@umdnj.edu

Journal of Sex & Marital Therapy
|April 25, 2007
PubMed
Summary
This summary is machine-generated.

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Trazodone discontinuation resolved ejaculatory inhibition in a male patient with psychiatric diagnoses. This highlights the importance of identifying specific psychotropic drug causes for sexual dysfunction.

Area of Science:

  • Psychiatry
  • Pharmacology
  • Sexual Medicine

Background:

  • Sexual adverse effects are frequently observed with psychotropic medications.
  • Polypharmacy in psychiatric treatment can complicate the identification of drug-induced side effects.
  • Co-morbid psychiatric diagnoses are common in patients requiring psychotropic therapy.

Observation:

  • A male patient with co-morbid psychiatric diagnoses experienced ejaculatory inhibition.
  • The patient was treated with a regimen including fluoxetine, divalproex sodium, lamotrigine, trazodone, and clonazepam.
  • A detailed timeline of psychotropic medication use and adverse effect onset was established.

Findings:

  • The onset of ejaculatory inhibition was temporally linked to the use of trazodone.

Related Experiment Videos

  • Discontinuation of trazodone led to the resolution of ejaculatory inhibition within 48 hours.
  • This case suggests trazodone as a potential cause of ejaculatory inhibition.

    • Implications:

    • Clinicians must be aware of trazodone's potential to cause ejaculatory inhibition.
    • Systematic and regular questioning is crucial for diagnosing sexual adverse effects of psychotropics.
    • Healthcare providers should be sensitive to potential patient reluctance, particularly religious patients, when discussing sexual side effects.