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Related Experiment Videos

Exposure, lung function decline and systemic inflammatory response in asphalt workers.

Bente Ulvestad1, Britt Grethe Randem, Siri Hetland

  • 1Mesta AS, PO Box 5133, N-1503 Moss, Norway. bente.ulvestad@mesta.no

Scandinavian Journal of Work, Environment & Health
|April 27, 2007
PubMed
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Asphalt paving work may lead to reduced lung function and increased inflammation markers like interleukin-6 (IL-6). This study highlights potential respiratory risks for asphalt workers during a work season.

Area of Science:

  • Occupational Health
  • Environmental Medicine
  • Pulmonary Medicine

Background:

  • Asphalt work involves exposure to various airborne contaminants.
  • Potential health effects of asphalt exposure on respiratory function and systemic inflammation are not fully understood.

Purpose of the Study:

  • To investigate the association between asphalt work exposures and changes in lung function.
  • To assess alterations in blood biomarkers, including interleukin-6 (IL-6), micro-C-reactive protein, and fibrinogen, among asphalt workers.

Main Methods:

  • Longitudinal study involving 140 asphalt workers and 126 heavy construction workers in Norway.
  • Spirometry performed pre- and post-season for asphalt workers; pre-season for controls.
  • Personal air sampling measured exposure to dust, oil mist, PAHs, and gases.

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Main Results:

  • Asphalt workers exhibited lower lung function (FEV1, FEF50) than controls at baseline.
  • Screedmen, a subgroup of pavers, showed significant declines in FVC and FEV1 post-season.
  • Mean plasma IL-6 increased significantly in asphalt pavers from 1.55 to 2.67 pg/ml (P=0.04).

Conclusions:

  • Asphalt paving exposure is linked to a potential risk of lung function decline.
  • Increased IL-6 suggests inflammatory responses associated with asphalt paving work.