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[Steroid responsive encephalopathy associated with auto-immune thyroiditis].

S Bannier1, F Taithe, C Solmon

  • 1Service de neurologie B, CHU Gabriel-Montpied, place Henri-Dunant, 63000 Clermont-Ferrand, France.

Annales D'Endocrinologie
|May 29, 2007
PubMed
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Hashimoto's encephalopathy, also known as steroid-responsive encephalopathy associated with autoimmune thyroiditis (SREAT), is a rare neurological condition. This study reviews diagnostic criteria, pathophysiology involving thyroid antibodies, and corticosteroid treatment options.

Area of Science:

  • Neurology
  • Immunology
  • Endocrinology

Background:

  • Hashimoto's thyroiditis is an autoimmune disease primarily affecting the thyroid gland.
  • Hashimoto's encephalopathy (HE) or SREAT is a rare neurological condition linked to autoimmune thyroid disease.
  • Over 100 cases of HE/SREAT have been reported since 1966.

Observation:

  • This report details two cases of HE/SREAT, highlighting variations in diagnostic approaches.
  • The cases provide an opportunity to examine pathophysiological hypotheses.
  • Key biological data, including serum and CSF antibodies (anti-TPO, anti-TG, anti-alpha-enolase), are discussed.

Findings:

  • The study emphasizes the role of specific autoantibodies in the pathophysiology of HE/SREAT.
  • Analysis of biological markers like anti-TPO, anti-TG, and anti-alpha-enolase antibodies is central to understanding the condition.

Related Experiment Videos

  • Diagnostic criteria and their implications are explored through the presented cases.

    • Implications:

    • Understanding the autoimmune basis of HE/SREAT is crucial for accurate diagnosis.
    • The findings support further investigation into the role of autoantibodies in neurological dysfunction.
    • Corticosteroid therapy remains a primary treatment option for HE/SREAT, with potential for improved patient outcomes.