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Pulmonary oedema complicating snake bite due to Bungarus caeruleus
R Agarwal1, A P Singh, A N Aggarwal
1Department of Pulmonary Medicine, Postgraduate Institute of Medical Education and Research, Sector 12, Chandigarh 160012, India. riteshpgi@gmail.com
Singapore Medical Journal
|July 28, 2007
Summary
Severe neurotoxic snakebites from Indian kraits can cause cardiotoxicity, leading to pulmonary edema. This case highlights the importance of recognizing cardiac complications in neurotoxic snake envenoming.
Area of Science:
- Toxicology
- Cardiology
- Herpetology
Background:
- Neurotoxic snake envenoming is a significant medical concern, particularly in regions with diverse venomous snake populations.
- While neurotoxicity is the primary concern with krait bites (Bungarus caeruleus), systemic effects can extend to other organ systems.
Observation:
- A 26-year-old Indian farmer presented with severe neurotoxic envenoming following a suspected Indian krait bite.
- The patient developed cardiogenic pulmonary edema, an unusual but critical cardiac manifestation.
Findings:
- This case represents the first reported instance of cardiotoxicity associated with Bungarus caeruleus envenoming.
- Literature review confirms that cardiac involvement, though rare, can occur in neurotoxic snakebites, presenting with various manifestations.
Implications:
- Early recognition of cardiac involvement in neurotoxic snake envenoming is crucial for timely and effective management.
- Anticipating complications like pulmonary edema allows for prompt medical intervention, improving patient outcomes in critical care settings.
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