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Virulence of non-beta-lactamase-mediated ampicillin-resistant Haemophilus influenzae

L G Rubin1, P M Mendelman, R M Rakita

  • 1Department of Pediatrics, Schneider Children's Hospital, Long Island Jewish Medical Center, New Hyde Park, NY 11042.

FEMS Microbiology Letters
|November 1, 1991
PubMed

Insights

Ampicillin-resistant Haemophilus influenzae with altered penicillin-binding proteins (PBPs) showed reduced virulence in infant rats. This suggests that changes in PBPs may impact bacterial growth and infection severity.

Area of Science:

  • Microbiology
  • Infectious Diseases
  • Bacterial Pathogenesis

Background:

  • Ampicillin resistance in Haemophilus influenzae is a growing concern.
  • Non-beta-lactamase-producing (AmpR NBLP) strains with altered penicillin-binding proteins (PBPs) are emerging.
  • The impact of altered PBPs on bacterial virulence is not well understood.

Purpose of the Study:

  • To investigate the virulence of ampicillin-resistant, non-beta-lactamase-producing Haemophilus influenzae strains with altered penicillin-binding proteins (PBPs).
  • To compare the virulence of these resistant strains against the susceptible recipient strain in a relevant animal model.

Main Methods:

  • Comparison of virulence using an infant rat model following intraperitoneal inoculation.
  • Assessment of mortality rates, bacteremia incidence and magnitude.
  • Evaluation of serum and neutrophil bactericidal activity and intravenous clearance rates.
  • In vitro growth studies in heated rat and human plasma.

Main Results:

  • Two of three ampicillin-resistant transformant strains exhibited significantly lower mortality and bacteremia compared to the susceptible recipient strain.
  • Reduced virulence was not linked to increased serum or neutrophil bactericidal activity or faster clearance.
  • Resistant transformants showed deficient in vivo multiplication, as evidenced by impaired growth in heated plasma.

Conclusions:

  • Haemophilus influenzae strains with altered PBPs demonstrate reduced virulence in the infant rat model.
  • The decreased virulence may be attributed to impaired in vivo multiplication.
  • These findings suggest a potential link between PBP alterations and reduced pathogenicity of H. influenzae.

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