Related Experiment Video
Updated: Aug 19, 2026

Robotic Myotomy and Partial Fundoplication for Achalasia
Published on: August 11, 2023
Involvement of the gastric antrum in Ménétrier's disease
Abstract:
Thirteen cases of Ménétrier's disease from the files of the Armed Forces Institute of Pathology that could be documented pathologically and correlated with appropriate roentgenograms were analyzed for the distribution of folds. Enlarged folds occurred in a localized area or in several areas simultaniously. The greater curvature of the body was most often involved (77 percent), but the antrum was affected in almost one-half (46 percent) of the cases; thus, the concept of relative antral sparing is not valid, and the diagnosis should be considered when there is gastric rugal enlargement in any distribution. The condition seems to be classified best as a hyperplastic gastropathy rather than a hypertrophic gastritis.
More Related Videos
Related Concept Videos
Pathophysiology of Peptic Ulcer Disease: Injurious Factors
In the antrum region, G cells secrete the gastrin hormone that binds to gastrin-cholecystokinin-B (CCK2) receptors on parietal and enterochromaffin-like (ECL) cells in the fundic glands. Simultaneously, the vagus nerve releases acetylcholine, which binds to M3...
Mucosal Barrier of the Stomach
Within parietal cells, carbonic acid is first formed through the reaction of water and carbon dioxide. The dissociation of carbonic acid releases bicarbonate and hydrogen ions. The bicarbonate...
Gastritis-II: Pathophysiology
In acute gastritis, the gastric mucosa becomes swollen and red and undergoes superficial erosion. Superficial ulceration may lead to bleeding.
In chronic gastritis, persistent or repeated insults lead to chronic inflammatory changes and, eventually, thinning or atrophy of the gastric tissue.
Gastritis can stem from various causes, each...
Peptic Ulcer Disease III: Clinical Manifestations and Diagnostic Studies
Few clinical manifestations differentiate gastric ulcers from duodenal ulcers. Distinctions in the location, timing, and pain relief are crucial for healthcare providers in differentiating between gastric and duodenal ulcers during clinical assessments.
Esophageal Achalasia
Gastritis II: Pathophysiology

