Related Experiment Video
Updated: Jul 11, 2026
07:57
Modeling Neonatal Intraventricular Hemorrhage Through Intraventricular Injection of Hemoglobin
Published on: August 25, 2022
Infected subgaleal hematoma in a neonate
Shirley Pollack1, Imad Kassis, Michalle Soudack
1Department of Neonatology, Meyer Children's Hospital, Israel.
The Pediatric Infectious Disease Journal
|September 13, 2007
Summary
Subgaleal hematoma (SGH) in newborns is rare but dangerous, potentially causing shock and death. This case highlights a severe, polymicrobial infection of SGH in a neonate, including unusual imaging findings.
Area of Science:
- Neonatal Medicine
- Pediatric Surgery
- Infectious Diseases
Background:
- Subgaleal hematoma (SGH) is a rare neonatal complication, often linked to vacuum-assisted deliveries.
- SGH can lead to severe anemia, hypovolemic shock, and mortality.
- Neonatal infected SGH is exceptionally uncommon, with only one prior reported case.
Observation:
- A term neonate presented with a complicated subgaleal hematoma.
- The neonate exhibited signs of severe infection involving multiple bacterial species.
- Unique imaging characteristics were noted in the affected SGH.
Findings:
- The subgaleal hematoma was complicated by a severe polymicrobial infection.
- Anaerobic bacteria were identified as part of the infectious agents.
- Distinctive imaging features differentiated this case from typical SGH presentations.
Implications:
- This case expands the understanding of SGH complications in neonates.
- Highlights the potential for severe, polymicrobial infections, including anaerobes, in SGH.
- Emphasizes the importance of considering infection in neonates with SGH and unique imaging findings.
Related Concept Videos
01:26Brain Abscess l: Introduction
A brain abscess is a focal, intracerebral infection characterized by a localized collection of pus within the brain parenchyma, resulting from microbial invasion and the body’s inflammatory response. It progresses through stages: early and late cerebritis, followed by early and late capsule formation, reflecting tissue destruction, immune response, and eventual encapsulation.Etiology and PathogenesisCausative organisms vary with source and host factors, often involving polymicrobial infections,...
01:29Hemorrhagic Stroke ll: Pathophysiology
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...
01:22Bacterial Meningitis I: Introduction
Bacterial meningitis is a severe, life-threatening inflammation of the meninges, particularly the pia mater and arachnoid mater, affecting the subarachnoid space, ventricles, and cerebrospinal fluid (CSF). If untreated, it can lead to significant neurological complications or death.Causative AgentsCommon pathogens vary with age and immune status. In adults, major organisms include Streptococcus pneumoniae, Neisseria meningitidis, and Haemophilus influenzae. Streptococcus agalactiae (group B...
01:17Hemorrhagic Stroke l: Introduction
A hemorrhagic stroke is an acute neurological event that occurs when a weakened cerebral blood vessel ruptures, allowing blood to accumulate within or around the brain. The sudden release of blood forms a focal hematoma that increases intracranial pressure, displaces neural tissue, and can obstruct cerebrospinal fluid pathways. These effects may be compounded by intraventricular extension of the hemorrhage, cerebral edema, or compression of adjacent structures, all of which contribute to...