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Left ventricular mass in elite olympic weight lifters
Sophie Lalande1, James Christopher Baldi
1Department of Sport and Exercise Science, University of Auckland, Auckland, New Zealand. slal028@ec.auckland.ac.nz
The American Journal of Cardiology
|September 22, 2007
Summary
Resistance training does not cause left ventricular (LV) hypertrophy. Elite weightlifters showed increased fat-free mass (FFM) but not LV mass, refuting hypertrophy hypotheses.
Area of Science:
- Cardiovascular Physiology
- Exercise Science
- Sports Medicine
Background:
- The relationship between resistance training and left ventricular (LV) concentric hypertrophy is debated.
- Previous studies show conflicting results regarding LV hypertrophy, potentially due to methodological limitations or confounding factors like steroid use.
Purpose of the Study:
- To investigate whether elite Olympic weightlifters exhibit resistance training-induced left ventricular concentric hypertrophy.
- To determine if LV structure and volumes differ between elite weightlifters and recreationally active controls.
Main Methods:
- Magnetic resonance imaging (MRI) was used to assess LV structure and volumes in 9 elite Olympic weightlifters and 10 controls.
- Body composition was determined using dual-energy x-ray absorptiometry (DXA).
- LV wall thickness was calculated from midventricular segment measurements.
Main Results:
- Weightlifters had similar age, weight, and fat-free mass (FFM) but were shorter and had a higher BMI than controls.
- No significant difference in LV mass was observed between groups; however, LV mass indexed to FFM was lower in weightlifters.
- LV mass correlated with FFM, but weightlifters had lower LV mass for a given FFM. LV wall thickness and volumes were not different.
Conclusions:
- The findings refute the hypothesis that resistance training induces LV concentric hypertrophy.
- Olympic weightlifting is associated with increased FFM without a corresponding increase in LV mass.
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