Related Experiment Video
Updated: Jul 11, 2026

04:02
System for Focal, Closed-System Central Nervous System Injury
Published on: November 29, 2024
Terson syndrome with bilateral optic nerve sheath hemorrhage
Chiaki D Gauntt1, Richard G Sherry, Chithra Kannan
1Department of Neurology, University of Louisville School of Medicine, Louisville, KY, USA. cgauntt@gaunttmd.com
Summary
Ruptured brain aneurysms can cause Terson syndrome, leading to vision loss. This case highlights CT findings of Terson syndrome with optic nerve sheath hemorrhage after an aneurysm rupture.
Area of Science:
- Neuro-ophthalmology
- Neuroradiology
- Vascular Neurology
Background:
- Subarachnoid hemorrhage (SAH) from anterior choroidal artery aneurysm rupture can lead to neurological and ophthalmological complications.
- Terson syndrome, characterized by intraocular hemorrhage following intracranial hemorrhage, is a known but infrequently reported association.
Observation:
- A 53-year-old male presented with acute headache and altered mental status.
- Preoperative CT revealed subarachnoid hemorrhage, bilateral optic nerve sheath hemorrhage, and bilateral intraocular hemorrhage.
- Ophthalmoscopy and B-scan ultrasound confirmed vitreous hemorrhages consistent with Terson syndrome.
Findings:
- This case presents the first computed tomography (CT) documentation of Terson syndrome manifesting with bilateral optic nerve sheath hemorrhage.
- The findings underscore the utility of CT in diagnosing the ophthalmological sequelae of aneurysmal SAH.
Implications:
- Early recognition of Terson syndrome is crucial for ophthalmological management and visual prognosis in patients with aneurysmal rupture.
- This report expands the neuroradiological understanding of Terson syndrome, emphasizing optic nerve sheath hemorrhage as a CT-detectable sign.
Related Concept Videos
Hepatic Encephalopathy
DefinitionHepatic encephalopathy is a reversible neurologic syndrome that results from advanced liver dysfunction or portosystemic shunting. It leads to disturbances in cognition, behavior, and motor function due to the brain’s exposure to gut-derived toxins that the liver fails to detoxify.EtiologyThis condition develops either in the setting of acute fulminant hepatitis or progressively during chronic liver disease, such as cirrhosis and portal hypertension. Portosystemic shunting—including...
Glaucoma: Overview
Glaucoma is an eye condition characterized by increased intraocular pressure that damages the retina and optic nerve, leading to irreversible blindness if left untreated. The human eye has various components, including the cornea, iris, pupil, lens, and optic nerve. Aqueous humor is secreted by the epithelium of the ciliary body in the posterior chamber and flows through the trabecular meshwork and canal of Schlemm, maintaining normal intraocular pressure. The trabecular meshwork and the canal...
Photoreceptors and Visual Pathways
At the molecular level, visual signals trigger transformations in photopigment molecules, resulting in changes in the photoreceptor cell's membrane potential. The photon's energy level is denoted by its wavelength, with each specific wavelength of visible light associated with a distinct color. The spectral range of visible light, classified as electromagnetic radiation, spans from 380 to 720 nm. Electromagnetic radiation wavelengths exceeding 720 nm fall under the infrared category, whereas...
Transient Ischemic Attack l: Introduction
A transient ischemic attack (TIA) is a brief episode of neurological dysfunction caused by a temporary, focal reduction in cerebral blood flow. Although symptoms resemble those of an ischemic stroke, the interruption in perfusion is short-lived and does not cause permanent infarction. TIAs are clinically important because they often serve as early warning events for future stroke.Mechanisms of Transient Cerebral IschemiaTransient cerebral ischemia may arise through several mechanisms. One...
Hemorrhagic Stroke ll: Pathophysiology
A hemorrhagic stroke develops when a cerebral blood vessel ruptures, allowing blood to escape into the surrounding brain tissue, as in intracerebral hemorrhage (ICH), or into the subarachnoid space, as in subarachnoid hemorrhage (SAH). Because the skull is a rigid compartment, the sudden presence of extravascular blood rapidly increases intracranial pressure and compresses adjacent neural structures, leading to immediate tissue injury and impaired cerebral perfusion.Mass Effect and Primary...