Cardiac denervation in postural tachycardia syndrome
Carl-Albrecht Haensch1, Hartmut Lerch, Anna Jigalin
1Department of Neurology, Autonomic Laboratory, HELIOS Klinikum Wuppertal, Heusnerstrabe 40, 42283 Wuppertal, Germany. carl-albrecht.haensch@helios-kliniken.de
Abstract:
Myocardial 123I-Meta-iodobenzylguanidine uptake was markedly reduced in a patient with postural tachycardia syndrome (POTS). This finding suggests that loss of sympathetic autonomic neurons in the heart may play a role in the etiology of POTS.
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