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Related Concept Videos

False Memories01:18

False Memories

False memories represent a cognitive distortion in which individuals recall events that did not happen, or remember them in an altered form. This phenomenon highlights the brain's constructive nature in processing and recalling memories, emphasizing that memory is not a perfect representation of past events but rather a dynamic reconstruction influenced by various factors.
One primary source of false memories is misattribution, where individuals incorrectly associate external information with...
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Related Experiment Video

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The Double-H Maze: A Robust Behavioral Test for Learning and Memory in Rodents
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Reduplicative paramnesia in Morvan's syndrome.

Lynsee A Hudson1, Yvonne D Rollins, C Alan Anderson

  • 1Department of Neurology, University of Colorado School of Medicine, Denver, Colorado, USA.

Journal of the Neurological Sciences
|October 12, 2007
PubMed
Summary
This summary is machine-generated.

Reduplicative paramnesia (RP) can occur in Morvan's syndrome associated with voltage-gated potassium channel antibodies (VGKC abs). Immunotherapy, including IV immunoglobulin and corticosteroids, may improve RP and nervous system hyperexcitability.

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Last Updated: Jul 11, 2026

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The Deese-Roediger-McDermott (DRM) Task: A Simple Cognitive Paradigm to Investigate False Memories in the Laboratory
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08:53

Using a Classroom-Based Deese Roediger McDermott Paradigm to Assess the Effects of Imagery on False Memories

Published on: November 14, 2018

Area of Science:

  • Neurology
  • Immunology

Background:

  • Morvan's syndrome involves peripheral nervous system hyperexcitability, including myokymia and neuromyotonia, often associated with VGKC abs.
  • Symptoms include hyperhydrosis, sleep disturbances, paresthesias, and encephalopathy.
  • Reduplicative paramnesia (RP) has not been previously reported in this condition.

Observation:

  • A 64-year-old male presented with a history of myokymia and myoclonus.
  • He experienced worsening parasomnia and confusion, developing a delusion of a duplicate home.
  • This presentation marked the first reported case of RP in Morvan's syndrome.

Findings:

  • Elevated serum VGKC antibody titers were detected.
  • Neuropsychological tests revealed executive function and memory deficits.
  • Electromyography confirmed diffuse myokymia. Treatment with IV immunoglobulin and prednisone improved RP and myoclonus, but not myokymia.

Implications:

  • RP is a potential neuropsychiatric manifestation of VGKC ab-associated Morvan's syndrome.
  • Both RP and nervous system hyperexcitability may respond to immunotherapy.
  • This case expands the understanding of Morvan's syndrome's clinical spectrum and treatment responses.