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Related Concept Videos

Synthesis and Regulation of Thyroid Hormones01:20

Synthesis and Regulation of Thyroid Hormones

Low blood levels of the thyroid hormones — triiodothyronine (T3) and thyroxine (T4) — signal the hypothalamus to release the thyrotropin-releasing hormone (TRH). TRH then reaches the pituitary gland and stimulates the release of thyroid-stimulating hormone(TSH) into the bloodstream.
Upon reaching the thyroid gland, TSH stimulates the follicular cells' active uptake of iodide ions from the blood. The ions diffuse to the apical surface of the cells and are oxidized to iodine. The iodine is then...
Hormones and Bone Tissue01:17

Hormones and Bone Tissue

The endocrine system produces and secretes hormones, which interact with the skeletal system. These hormones control bone growth, maintain bone once it is formed, and remodel it.
Hormones That Influence Osteoblasts and/or Maintain the Matrix
Several hormones are necessary for controlling bone growth and maintaining the bone matrix. The pituitary gland secretes growth hormone (GH), which, as its name implies, controls bone growth. This happens in several ways: first, it triggers chondrocyte...
Functions of Thyroid Hormones01:18

Functions of Thyroid Hormones

The thyroid hormone (TH) plays a pivotal role in the intricate orchestration of physiological processes, exerting profound effects on development, metabolism, and homeostasis throughout different life stages.
TH is indispensable for the normal development and maturation of the skeletal, muscular, and nervous systems during fetal and childhood growth. It facilitates bone mineral turnover and regulates protein synthesis in developing tissues, contributing significantly to overall growth and...
Hypothyroidism II: Pathophysiology01:23

Hypothyroidism II: Pathophysiology

Hypothyroidism is a disorder characterized by insufficient production of thyroid hormones, which regulate metabolism, energy balance, and multiple organ systems.TypesHypothyroidism is classified based on the level of dysfunction. Primary hypothyroidism results from intrinsic thyroid gland dysfunction, causing reduced hormone production despite normal or increased stimulation. Secondary hypothyroidism arises from inadequate thyroid-stimulating hormone (TSH) secretion by the pituitary. Tertiary...
Hyperthyroidism II: Pathophysiology01:27

Hyperthyroidism II: Pathophysiology

Hyperthyroidism is a hypermetabolic state caused by elevated levels of thyroid hormones, triiodothyronine (T3) and thyroxine (T4). It results from dysregulation at the thyroid, pituitary, or immune system level and affects multiple organ systems.PathophysiologyThe most common cause of hyperthyroidism is Graves’ disease, an autoimmune disorder in which antibodies, specifically thyroid-stimulating antibodies (TSAb), a subtype of TSH receptor antibodies (TRAb), bind to and activate TSH receptors...
Hyperthyroidism I: Introduction01:25

Hyperthyroidism I: Introduction

Hyperthyroidism is a type of thyrotoxicosis characterized by the thyroid gland's overproduction of the thyroid hormones triiodothyronine (T3) and thyroxine (T4). This hormone excess increases the basal metabolic rate and enhances sensitivity to catecholamines.DiagnosisDiagnosis is based on clinical features and biochemical testing. It typically shows suppressed thyroid-stimulating hormone (TSH) levels below 0.4 mIU/L, with elevated free T3 and/or T4. Additional tests, including thyroid...

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Related Experiment Video

Updated: Jul 10, 2026

Synchronous Triplanar Reconstruction Integrated with Color Doppler Mapping for Precise and Rapid Localization of Thyroid Lesions
05:41

Synchronous Triplanar Reconstruction Integrated with Color Doppler Mapping for Precise and Rapid Localization of Thyroid Lesions

Published on: February 9, 2024

Does diagnostic sonography alter thyroid and parathyroid hormone levels?

Erdinc Serin1, Kamil Gurel, Aysu Kiyan

  • 1Department of Biochemistry, Abant Izzet Baysal University, Izzet Baysal Medical Faculty, Golkoy, Bolu, 14280 Turkey.

Journal of Clinical Ultrasound : JCU
|October 17, 2007
PubMed
Summary

Diagnostic ultrasound of the thyroid may alter parathyroid hormone (PTH) levels. This study found significant PTH variations after both sonographic examination and probe-only application, suggesting external factors can influence hormone levels.

Related Experiment Videos

Last Updated: Jul 10, 2026

Synchronous Triplanar Reconstruction Integrated with Color Doppler Mapping for Precise and Rapid Localization of Thyroid Lesions
05:41

Synchronous Triplanar Reconstruction Integrated with Color Doppler Mapping for Precise and Rapid Localization of Thyroid Lesions

Published on: February 9, 2024

Area of Science:

  • Endocrinology
  • Medical Imaging

Background:

  • Thyroid sonography is a common diagnostic tool.
  • Potential physiological effects of ultrasound on endocrine function are not fully understood.

Purpose of the Study:

  • To evaluate changes in thyroid and parathyroid hormone (PTH) and thyroglobulin (TG) levels in healthy individuals after diagnostic thyroid ultrasound.
  • To determine if ultrasound examination influences serum hormone concentrations.

Main Methods:

  • Thirty healthy women underwent two examinations: diagnostic ultrasound and a probe-only application (control).
  • Blood samples were collected before and after each examination to measure thyroid hormones, PTH, and TG.
  • Variations in hormone levels between the two examination types were compared.

Main Results:

  • Significant increases in thyroglobulin (TG) were observed after the probe-only application.
  • Parathyroid hormone (PTH) levels showed significant variations after both the diagnostic ultrasound and the probe-only application.
  • The difference in variations between the two applications was significant only for PTH.

Conclusions:

  • Thyroid sonography and related procedures may influence serum parathyroid hormone (PTH) levels.
  • External factors, including ultrasound, can potentially affect endocrine gland secretions.
  • Clinicians should consider potential hormone level alterations due to external influences.