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Related Concept Videos

Graves Disease II: Pathophysiology01:24

Graves Disease II: Pathophysiology

Graves’ disease is an autoimmune disorder characterized by the production of thyroid-stimulating immunoglobulins (TSI) that activate TSH receptors, leading to excessive synthesis and release of thyroid hormones (T3 and T4) and resulting in hyperthyroidism.Among all causes of hyperthyroidism, Graves’ disease is the most common and can happen at any age, though it is more frequent in women. It produces a hypermetabolic state with features such as weight loss, tachycardia, tremor, and heat...
Graves' Disease I: Introduction01:28

Graves' Disease I: Introduction

Graves' disease is an autoimmune disorder that causes hyperthyroidism, or overactivity of the thyroid gland. It results from autoantibodies called thyroid-stimulating immunoglobulins (TSIs), which bind to thyroid-stimulating hormone (TSH) receptors, leading to overstimulation of hormone production and a hypermetabolic state.EtiologyAlthough considered idiopathic, Graves’ disease has well-established contributing factors. There is a strong genetic component, with increased prevalence in...
Tonsillitis I: Introduction01:30

Tonsillitis I: Introduction

Tonsillitis is inflammation of the tonsils, which are two lymphoid tissue masses at the back of the throat. This condition can cause discomfort and irritation in the throat.
Etiology
Three primary contributing factors have been identified.
Hyperthyroidism II: Pathophysiology01:27

Hyperthyroidism II: Pathophysiology

Hyperthyroidism is a hypermetabolic state caused by elevated levels of thyroid hormones, triiodothyronine (T3) and thyroxine (T4). It results from dysregulation at the thyroid, pituitary, or immune system level and affects multiple organ systems.PathophysiologyThe most common cause of hyperthyroidism is Graves’ disease, an autoimmune disorder in which antibodies, specifically thyroid-stimulating antibodies (TSAb), a subtype of TSH receptor antibodies (TRAb), bind to and activate TSH receptors...
Tooth Anatomy01:21

Tooth Anatomy

The human tooth enables us to eat a variety of foods, speak clearly, and even aid in shaping our faces. Teeth are composed of various elements that work together. Here's a detailed look at the anatomy of a human tooth.
The Crown, Neck, and Root
The visible part of the tooth is referred to as the crown. It's covered by enamel, the hardest substance in the human body. The crown is uniquely shaped for each type of tooth, allowing for different functions such as cutting, tearing, or grinding food.
Hyperthyroidism I: Introduction01:25

Hyperthyroidism I: Introduction

Hyperthyroidism is a type of thyrotoxicosis characterized by the thyroid gland's overproduction of the thyroid hormones triiodothyronine (T3) and thyroxine (T4). This hormone excess increases the basal metabolic rate and enhances sensitivity to catecholamines.DiagnosisDiagnosis is based on clinical features and biochemical testing. It typically shows suppressed thyroid-stimulating hormone (TSH) levels below 0.4 mIU/L, with elevated free T3 and/or T4. Additional tests, including thyroid...

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Related Experiment Video

Updated: Jul 10, 2026

Lateral Molar Approach-Driven Transoral Endoscopic Procedure for Benign Infratemporal Fossa Tumor Resection
04:04

Lateral Molar Approach-Driven Transoral Endoscopic Procedure for Benign Infratemporal Fossa Tumor Resection

Published on: August 15, 2025

Acute thyroiditis of odontogenic origin.

A Acocella1, P Nardi, R Sacco

  • 1Department of Odontostomatology, Division of Maxillo-Facial Surgery, Faculty of Medicine, University of Florence, Florence, Italy. alessandroacocella@yahoo.it

Minerva Stomatologica
|October 17, 2007
PubMed
Summary

A dental abscess can spread to the neck and thyroid gland, causing thyrotoxicosis. Prompt dental and medical treatment resolved the condition and prevented recurrence.

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Last Updated: Jul 10, 2026

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Area of Science:

  • Endocrinology
  • Oral Surgery
  • Infectious Disease

Background:

  • Dental abscesses can spread through fascial planes if not properly drained.
  • Untreated infections may descend into the submandibular, lateral pharyngeal, and retro-pharyngeal spaces.
  • The thyroid gland is a potential site for infection spread from deep neck abscesses.

Observation:

  • A case of submandibular phlegmon originating from a periapical abscess of an inferior premolar is presented.
  • The infection extended to the thyroid gland, causing significant damage.
  • Thyroid gland edema led to dysphagia and dysphonia.

Findings:

  • Thyroid gland damage resulted in the release of excess thyroid hormones, manifesting as thyrotoxicosis.
  • Symptoms included temperature, pallor, perspiration, tremor, fatigue, weight loss, increased appetite, and tachycardia.
  • The patient experienced dysphagia and dysphonia due to thyroid gland edema.

Implications:

  • This case highlights the critical importance of early dental abscess drainage to prevent severe systemic complications.
  • Effective management requires a multidisciplinary approach involving dental, endocrine, and infectious disease specialists.
  • Prompt treatment of thyrotoxicosis and the underlying dental infection is crucial for patient recovery and preventing recurrence.